Downregulation of ATP1A1 promotes cancer development in renal cell carcinoma
Published 2017 View Full Article
- Home
- Publications
- Publication Search
- Publication Details
Title
Downregulation of ATP1A1 promotes cancer development in renal cell carcinoma
Authors
Keywords
Na<sup>+</sup>/K<sup>+</sup>-ATPase α1 subunit, Downregulation, Renal cell carcinoma
Journal
Clinical Proteomics
Volume 14, Issue 1, Pages -
Publisher
Springer Nature
Online
2017-05-04
DOI
10.1186/s12014-017-9150-4
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- Downregulation of ARHGDIA contributes to human glioma progression through activation of Rho GTPase signaling pathway
- (2016) Weiliang Lu et al. TUMOR BIOLOGY
- Na+/K+-ATPase α1 subunit, a novel therapeutic target for hepatocellular carcinoma
- (2015) Liping Zhuang et al. Oncotarget
- The Overexpression of IQGAP1 and β-Catenin Is Associated with Tumor Progression in Hepatocellular Carcinoma In Vitro and In Vivo
- (2015) Xuewen Jin et al. PLoS One
- Epigenetic silencing of Na,K-ATPase β1subunit geneATP1B1by methylation in clear cell renal cell carcinoma
- (2014) Ponniah Selvakumar et al. Epigenetics
- The Na-K-ATPase α1β1 heterodimer as a cell adhesion molecule in epithelia
- (2012) Olga Vagin et al. AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY
- Quantitative Proteomics for Cancer Biomarker Discovery
- (2012) Shufang Liang et al. COMBINATORIAL CHEMISTRY & HIGH THROUGHPUT SCREENING
- Na/K-ATPase Mimetic pNaKtide Peptide Inhibits the Growth of Human Cancer Cells
- (2011) Zhichuan Li et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Protein profiling of microdomains purified from renal cell carcinoma and normal kidney tissue samples
- (2011) F. Raimondo et al. Molecular BioSystems
- Use of stable isotope labeling by amino acids in cell culture as a spike-in standard in quantitative proteomics
- (2011) Tamar Geiger et al. Nature Protocols
- A HIF-Regulated VHL-PTP1B-Src Signaling Axis Identifies a Therapeutic Target in Renal Cell Carcinoma
- (2011) N. Suwaki et al. Science Translational Medicine
- ROS induced DNA damage and checkpoint responses: Influences on aging?
- (2010) Luis Miguel Guachalla et al. CELL CYCLE
- Application of the SILAC (stable isotope labelling with amino acids in cell culture) technique in quantitative comparisons for tissue proteome expression
- (2009) Yuhuan Xu et al. BIOTECHNOLOGY AND APPLIED BIOCHEMISTRY
- Quantitative protein expression profiling of 14-3-3 isoforms in human renal carcinoma shows 14-3-3 epsilon is involved in limitedly increasing renal cell proliferation
- (2009) Shufang Liang et al. ELECTROPHORESIS
- Na,K-ATPase and epithelial tight junctions
- (2009) Sigrid et al. Frontiers in Bioscience-Landmark
- The sodium pump α1 sub-unit: a disease progression-related target for metastatic melanoma treatment
- (2009) Véronique Mathieu et al. JOURNAL OF CELLULAR AND MOLECULAR MEDICINE
- Resistance to targeted therapy in renal-cell carcinoma
- (2009) Brian I Rini et al. LANCET ONCOLOGY
- The DNA-damage response in human biology and disease
- (2009) Stephen P. Jackson et al. NATURE
- TARGETING THE α 1 SUBUNIT OF THE SODIUM PUMP TO COMBAT GLIOBLASTOMA CELLS
- (2009) Florence Lefranc et al. NEUROSURGERY
- Isoform-specific expression and characterization of 14-3-3 proteins in human glioma tissues discovered by stable isotope labeling with amino acids in cell culture-based proteomic analysis
- (2009) Shufang Liang et al. Proteomics Clinical Applications
- Gene expression and methylation status of 14‐3‐3σ in human renal carcinoma tissues
- (2008) Shufang Liang et al. IUBMB LIFE
- Na,K-Adenosine Triphosphatase α1-Subunit Predicts Survival of Renal Clear Cell Carcinoma
- (2007) David B. Seligson et al. JOURNAL OF UROLOGY
Find Funding. Review Successful Grants.
Explore over 25,000 new funding opportunities and over 6,000,000 successful grants.
ExplorePublish scientific posters with Peeref
Peeref publishes scientific posters from all research disciplines. Our Diamond Open Access policy means free access to content and no publication fees for authors.
Learn More