Curcumin Activates AMPK Pathway and Regulates Lipid Metabolism in Rats Following Prolonged Clozapine Exposure
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Title
Curcumin Activates AMPK Pathway and Regulates Lipid Metabolism in Rats Following Prolonged Clozapine Exposure
Authors
Keywords
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Journal
Frontiers in Neuroscience
Volume 11, Issue -, Pages -
Publisher
Frontiers Media SA
Online
2017-10-04
DOI
10.3389/fnins.2017.00558
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Note: Only part of the references are listed.- Curcumin as a potential candidate for treating hyperlipidemia: A review of cellular and metabolic mechanisms
- (2017) Yunes Panahi et al. JOURNAL OF CELLULAR PHYSIOLOGY
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- (2017) Jaewoong Jang et al. Molecular Medicine Reports
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- (2017) Jia Choi et al. Nutrients
- Therapeutic efficacy of atypical antipsychotic drugs by targeting multiple stress-related metabolic pathways
- (2017) H L Cai et al. Translational Psychiatry
- Curcumin induces brown fat-like phenotype in 3T3-L1 and primary white adipocytes
- (2016) Jameel Lone et al. JOURNAL OF NUTRITIONAL BIOCHEMISTRY
- Curcumin inhibits hyperlipidemia and hepatic fat accumulation in high-fructose-fed male Wistar rats
- (2016) Nachimuthu Maithilikarpagaselvi et al. PHARMACEUTICAL BIOLOGY
- Ameliorating antipsychotic-induced weight gain by betahistine: Mechanisms and clinical implications
- (2016) Jiamei Lian et al. PHARMACOLOGICAL RESEARCH
- Curcumin rescues high fat diet-induced obesity and insulin sensitivity in mice through regulating SREBP pathway
- (2016) Lili Ding et al. TOXICOLOGY AND APPLIED PHARMACOLOGY
- Curcumin suppresses colon cancer cell invasion via AMPK-induced inhibition of NF–κB, uPA activator and MMP9
- (2016) Weihua Tong et al. Oncology Letters
- Clozapine modifies the differentiation program of human adipocytes inducing browning
- (2016) E Kristóf et al. Translational Psychiatry
- Vitamin D deficiency exacerbates atypical antipsychotic-induced metabolic side effects in rats: Involvement of the INSIG/SREBP pathway
- (2015) Ruili Dang et al. EUROPEAN NEUROPSYCHOPHARMACOLOGY
- Olanzapine depot exposure in male rats: Dose-dependent lipogenic effects without concomitant weight gain
- (2015) J. Fernø et al. EUROPEAN NEUROPSYCHOPHARMACOLOGY
- Betahistine co-treatment ameliorates dyslipidemia induced by chronic olanzapine treatment in rats through modulation of hepatic AMPKα-SREBP-1 and PPARα-dependent pathways
- (2015) Xuemei Liu et al. PHARMACOLOGICAL RESEARCH
- A potential mechanism underlying atypical antipsychotics-induced lipid disturbances
- (2015) H L Cai et al. Translational Psychiatry
- AMPK activation prevents excess nutrient-induced hepatic lipid accumulation by inhibiting mTORC1 signaling and endoplasmic reticulum stress response
- (2014) Hongliang Li et al. BIOCHIMICA ET BIOPHYSICA ACTA-MOLECULAR BASIS OF DISEASE
- Olanzapine-Activated AMPK Signaling in the Dorsal Vagal Complex Is Attenuated by Histamine H1 Receptor Agonist in Female Rats
- (2014) Meng He et al. ENDOCRINOLOGY
- Effect of tree nuts on metabolic syndrome criteria: a systematic review and meta-analysis of randomised controlled trials
- (2014) S. Blanco Mejia et al. BMJ Open
- Atypical antipsychotics alter cholesterol and fatty acid metabolism in vitro
- (2012) Alberto Canfrán-Duque et al. JOURNAL OF LIPID RESEARCH
- Curcumin decreases renal triglyceride accumulation through AMPK–SREBP signaling pathway in streptozotocin-induced type 1 diabetic rats
- (2012) Vivian Soetikno et al. JOURNAL OF NUTRITIONAL BIOCHEMISTRY
- Chronic olanzapine administration in rats: Effect of route of administration on weight, food intake and body composition
- (2012) Steve Mann et al. PHARMACOLOGY BIOCHEMISTRY AND BEHAVIOR
- Curcumin Prevents High Fat Diet Induced Insulin Resistance and Obesity via Attenuating Lipogenesis in Liver and Inflammatory Pathway in Adipocytes
- (2012) Weijuan Shao et al. PLoS One
- AMPK functions as an adenylate charge-regulated protein kinase
- (2012) Jonathan S. Oakhill et al. TRENDS IN ENDOCRINOLOGY AND METABOLISM
- Atypical antipsychotic drugs perturb AMPK-dependent regulation of hepatic lipid metabolism
- (2011) Kyoung-Jin Oh et al. AMERICAN JOURNAL OF PHYSIOLOGY-ENDOCRINOLOGY AND METABOLISM
- Olanzapine, but not aripiprazole, weight-independently elevates serum triglycerides and activates lipogenic gene expression in female rats
- (2011) Silje Skrede et al. INTERNATIONAL JOURNAL OF NEUROPSYCHOPHARMACOLOGY
- Does endoplasmic reticulum stress participate in APD-induced hepatic metabolic dysregulation?
- (2011) Emilie Lauressergues et al. NEUROPHARMACOLOGY
- Atypical antipsychotic medications increase postprandial triglyceride and glucose levels in male rats: Relationship with stearoyl-CoA desaturase activity
- (2011) Robert K. McNamara et al. SCHIZOPHRENIA RESEARCH
- Antipsychotic drug action on SREBPs-related lipogenesis and cholesterogenesis in primary rat hepatocytes
- (2010) Emilie Lauressergues et al. NAUNYN-SCHMIEDEBERGS ARCHIVES OF PHARMACOLOGY
- Curcumin Inhibitssrebp-2Expression in Activated Hepatic Stellate Cellsin Vitroby Reducing the Activity of Specificity Protein-1
- (2009) Qiaohua Kang et al. ENDOCRINOLOGY
- Curcumin Exerts Antidifferentiation Effect through AMPKα-PPAR-γ in 3T3-L1 Adipocytes and Antiproliferatory Effect through AMPKα-COX-2 in Cancer Cells
- (2008) Yun K. Lee et al. JOURNAL OF AGRICULTURAL AND FOOD CHEMISTRY
- Investigation into the influence of a high fat diet on antipsychotic-induced weight gain in female rats
- (2008) MJ Fell et al. JOURNAL OF PSYCHOPHARMACOLOGY
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