Phloretin ameliorates arsenic trioxide induced mitochondrial dysfunction in H9c2 cardiomyoblasts mediated via alterations in membrane permeability and ETC complexes
Phloretin ameliorates arsenic trioxide induced mitochondrial dysfunction in H9c2 cardiomyoblasts mediated via alterations in membrane permeability and ETC complexes
Authors
Keywords
Arsenic trioxide, Phloretin, H9c2 cells, Electron transport chain, Mitochondrial swelling, Oxygen consumption
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