Loss of receptor activity-modifying protein 2 in mice causes placental dysfunction and alters PTH1R regulation
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Title
Loss of receptor activity-modifying protein 2 in mice causes placental dysfunction and alters PTH1R regulation
Authors
Keywords
Placenta, Trophoblasts, Parathyroid hormone, G protein coupled receptors, Immunofluorescence, Arteries, Mouse models, Gene expression
Journal
PLoS One
Volume 12, Issue 7, Pages e0181597
Publisher
Public Library of Science (PLoS)
Online
2017-07-21
DOI
10.1371/journal.pone.0181597
References
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- Research Resource: Haploinsufficiency of Receptor Activity-Modifying Protein-2 (Ramp2) Causes Reduced Fertility, Hyperprolactinemia, Skeletal Abnormalities, and Endocrine Dysfunction in Mice
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- Parathyroid Carcinoma: Current Understanding and New Insights into Gene Expression and Intraoperative Parathyroid Hormone Kinetics
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- Parathyroid hormone regulates fetal-placental mineral homeostasis
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- Adrenomedullin signaling is necessary for murine lymphatic vascular development
- (2007) Kimberly L. Fritz-Six et al. JOURNAL OF CLINICAL INVESTIGATION
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