Vinpocetine reduces diclofenac-induced acute kidney injury through inhibition of oxidative stress, apoptosis, cytokine production, and NF-κB activation in mice
Vinpocetine reduces diclofenac-induced acute kidney injury through inhibition of oxidative stress, apoptosis, cytokine production, and NF-κB activation in mice
Authors
Keywords
Diclofenac, Acute kidney injury, NF-κB, Vinpocetine, Proteinuria, Reactive oxygen species
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