Arid1b haploinsufficiency disrupts cortical interneuron development and mouse behavior
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Title
Arid1b haploinsufficiency disrupts cortical interneuron development and mouse behavior
Authors
Keywords
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Journal
NATURE NEUROSCIENCE
Volume 20, Issue 12, Pages 1694-1707
Publisher
Springer Nature
Online
2017-11-04
DOI
10.1038/s41593-017-0013-0
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- (2015) Jessica Mariani et al. CELL
- Loss of GSK-3 Causes Abnormal Astrogenesis and Behavior in Mice
- (2015) Eui-Man Jung et al. MOLECULAR NEUROBIOLOGY
- Microtubule-Actin Crosslinking Factor 1 Is Required for Dendritic Arborization and Axon Outgrowth in the Developing Brain
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- (2015) Sacha B. Nelson et al. NEURON
- Role of nucleosome remodeling in neurodevelopmental and intellectual disability disorders
- (2015) Alberto J. López et al. Frontiers in Behavioral Neuroscience
- Genes and brain malformations associated with abnormal neuron positioning
- (2015) Jeffrey J. Moffat et al. Molecular Brain
- Lack of parvalbumin in mice leads to behavioral deficits relevant to all human autism core symptoms and related neural morphofunctional abnormalities
- (2015) M Wöhr et al. Translational Psychiatry
- mTOR regulates brain morphogenesis by mediating GSK3 signaling
- (2014) M. Ka et al. DEVELOPMENT
- MACF1 regulates the migration of pyramidal neurons via microtubule dynamics and GSK-3 signaling
- (2014) Minhan Ka et al. DEVELOPMENTAL BIOLOGY
- PGC-1 Provides a Transcriptional Framework for Synchronous Neurotransmitter Release from Parvalbumin-Positive Interneurons
- (2014) E. K. Lucas et al. JOURNAL OF NEUROSCIENCE
- The GABA excitatory/inhibitory developmental sequence: A personal journey
- (2014) Y. Ben-Ari NEUROSCIENCE
- Patches of Disorganization in the Neocortex of Children with Autism
- (2014) Rich Stoner et al. NEW ENGLAND JOURNAL OF MEDICINE
- GABAergic Signaling as Therapeutic Target for Autism Spectrum Disorders
- (2014) Giada Cellot et al. Frontiers in Pediatrics
- Knockout of NMDA Receptors in Parvalbumin Interneurons Recreates Autism-Like Phenotypes
- (2013) John A. Saunders et al. Autism Research
- Altered neural connectivity in excitatory and inhibitory cortical circuits in autism
- (2013) Basilis Zikopoulos et al. Frontiers in Human Neuroscience
- The neuron-specific chromatin regulatory subunit BAF53b is necessary for synaptic plasticity and memory
- (2013) Annie Vogel-Ciernia et al. NATURE NEUROSCIENCE
- From neural development to cognition: unexpected roles for chromatin
- (2013) Jehnna L. Ronan et al. NATURE REVIEWS GENETICS
- Haploinsufficiency of ARID1B , a Member of the SWI/SNF-A Chromatin-Remodeling Complex, Is a Frequent Cause of Intellectual Disability
- (2012) Juliane Hoyer et al. AMERICAN JOURNAL OF HUMAN GENETICS
- Genetic Basis of Intellectual Disability
- (2012) Jay W. Ellison et al. Annual Review of Medicine
- Autistic-like behaviour in Scn1a +/− mice and rescue by enhanced GABA-mediated neurotransmission
- (2012) Sung Han et al. NATURE
- Mutations in SWI/SNF chromatin remodeling complex gene ARID1B cause Coffin-Siris syndrome
- (2012) Gijs W E Santen et al. NATURE GENETICS
- Mutations affecting components of the SWI/SNF complex cause Coffin-Siris syndrome
- (2012) Yoshinori Tsurusaki et al. NATURE GENETICS
- Interneuron dysfunction in psychiatric disorders
- (2012) Oscar Marín NATURE REVIEWS NEUROSCIENCE
- Correction: Synaptotagmin-2 Is a Reliable Marker for Parvalbumin Positive Inhibitory Boutons in the Mouse Visual Cortex
- (2012) Jean-Pierre Sommeijer et al. PLoS One
- Corpus callosum abnormalities, intellectual disability, speech impairment, and autism in patients with haploinsufficiency of ARID1B
- (2011) C Halgren et al. CLINICAL GENETICS
- Apoptosis- and endoplasmic reticulum stress-related genes were regulated by estrogen and progesterone in the uteri of calbindin-D9k and -D28k knockout mice
- (2011) Eui-Man Jung et al. JOURNAL OF CELLULAR BIOCHEMISTRY
- Managing anxiety and depressive symptoms in adults with autism-spectrum disorders
- (2011) Kerry Boyd JOURNAL OF PSYCHIATRY & NEUROSCIENCE
- A conditional knockout resource for the genome-wide study of mouse gene function
- (2011) William C. Skarnes et al. NATURE
- Histone H3 tail acetylation modulates ATP-dependent remodeling through multiple mechanisms
- (2011) Nilanjana Chatterjee et al. NUCLEIC ACIDS RESEARCH
- Parvalbumin-, calbindin-, and calretinin-immunoreactive hippocampal interneuron density in autism
- (2009) Y. A. Lawrence et al. ACTA NEUROLOGICA SCANDINAVICA
- Common circuit defect of excitatory-inhibitory balance in mouse models of autism
- (2009) Nadine Gogolla et al. Journal of Neurodevelopmental Disorders
- The SWI/SNF chromatin remodeling subunit BRG1 is a critical regulator of p53 necessary for proliferation of malignant cells
- (2009) S R Naidu et al. ONCOGENE
- Fate mapping Nkx2.1-lineage cells in the mouse telencephalon
- (2007) Qing Xu et al. JOURNAL OF COMPARATIVE NEUROLOGY
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