Withdrawal from Chronic Alcohol Induces a Unique CCL2 mRNA Increase in Adolescent But Not Adult Brain-Relationship to Blood Alcohol Levels and Seizures
Published 2015 View Full Article
- Home
- Publications
- Publication Search
- Publication Details
Title
Withdrawal from Chronic Alcohol Induces a Unique CCL2 mRNA Increase in Adolescent But Not Adult Brain-Relationship to Blood Alcohol Levels and Seizures
Authors
Keywords
-
Journal
ALCOHOLISM-CLINICAL AND EXPERIMENTAL RESEARCH
Volume 39, Issue 12, Pages 2375-2385
Publisher
Wiley
Online
2015-11-11
DOI
10.1111/acer.12898
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- Neuroimmune Activation and Myelin Changes in Adolescent Rats Exposed to High-Dose Alcohol and Associated Cognitive Dysfunction: A Review with Reference to Human Adolescent Drinking
- (2013) María Pascual et al. ALCOHOL AND ALCOHOLISM
- The Cytokine mRNA Increase Induced by Withdrawal from Chronic Ethanol in the Sterile Environment of Brain is Mediated by CRF and HMGB1 Release
- (2013) Buddy A. Whitman et al. ALCOHOLISM-CLINICAL AND EXPERIMENTAL RESEARCH
- Effects of Ethanol on Immune Response in the Brain: Region-Specific Changes in Adolescent Versus Adult Mice
- (2013) Cynthia J. M. Kane et al. ALCOHOLISM-CLINICAL AND EXPERIMENTAL RESEARCH
- Opposing actions of hippocampus TNFα receptors on limbic seizure susceptibility
- (2013) Marc S. Weinberg et al. EXPERIMENTAL NEUROLOGY
- Age of first drinking and adult alcohol problems: systematic review of prospective cohort studies
- (2013) Will Maimaris et al. JOURNAL OF EPIDEMIOLOGY AND COMMUNITY HEALTH
- Monocyte chemoattractant protein-1 affects migration of hippocampal neural progenitors following status epilepticus in rats
- (2013) Yu-Wen Hung et al. Journal of Neuroinflammation
- Increased receptor for advanced glycation end product expression in the human alcoholic prefrontal cortex is linked to adolescent drinking
- (2013) Ryan P. Vetreno et al. NEUROBIOLOGY OF DISEASE
- High Mobility Group Box 1/Toll-like Receptor Danger Signaling Increases Brain Neuroimmune Activation in Alcohol Dependence
- (2012) Fulton T. Crews et al. BIOLOGICAL PSYCHIATRY
- Temporal changes in innate immune signals in a rat model of alcohol withdrawal in emotional and cardiorespiratory homeostatic nuclei
- (2012) Kate Freeman et al. Journal of Neuroinflammation
- Cytokine involvement in stress may depend on corticotrophin releasing factor to sensitize ethanol withdrawal anxiety
- (2011) Darin J. Knapp et al. BRAIN BEHAVIOR AND IMMUNITY
- Epileptogenesis Provoked by Prolonged Experimental Febrile Seizures: Mechanisms and Biomarkers
- (2010) C. M. Dube et al. JOURNAL OF NEUROSCIENCE
- The role of inflammation in epilepsy
- (2010) Annamaria Vezzani et al. Nature Reviews Neurology
- Chemokine CCL2 and its receptor CCR2 are increased in the hippocampus following pilocarpine-induced status epilepticus
- (2009) Maira L Foresti et al. Journal of Neuroinflammation
- Cellular injury and neuroinflammation in children with chronic intractable epilepsy
- (2009) Jieun Choi et al. Journal of Neuroinflammation
- Differential Dietary Ethanol Intake and Blood Ethanol Levels in Adolescent and Adult Rats: Effects on Anxiety-Like Behavior and Seizure Thresholds
- (2008) Tiffany A. Wills et al. ALCOHOLISM-CLINICAL AND EXPERIMENTAL RESEARCH
- Sensitization, Duration, and Pharmacological Blockade of Anxiety-Like Behavior Following Repeated Ethanol Withdrawal in Adolescent and Adult Rats
- (2008) Tiffany A. Wills et al. ALCOHOLISM-CLINICAL AND EXPERIMENTAL RESEARCH
- Increased MCP-1 and microglia in various regions of the human alcoholic brain
- (2007) Jun He et al. EXPERIMENTAL NEUROLOGY
Publish scientific posters with Peeref
Peeref publishes scientific posters from all research disciplines. Our Diamond Open Access policy means free access to content and no publication fees for authors.
Learn MoreAsk a Question. Answer a Question.
Quickly pose questions to the entire community. Debate answers and get clarity on the most important issues facing researchers.
Get Started