Compensatory axon sprouting for very slow axonal die-back in a transgenic model of spinal muscular atrophy type III
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Title
Compensatory axon sprouting for very slow axonal die-back in a transgenic model of spinal muscular atrophy type III
Authors
Keywords
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Journal
JOURNAL OF PHYSIOLOGY-LONDON
Volume 595, Issue 5, Pages 1815-1829
Publisher
Wiley
Online
2016-11-28
DOI
10.1113/jp273404
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Note: Only part of the references are listed.- Characterization of Neuronal Death and Functional Deficits following Nerve Injury during the Early Postnatal Developmental Period in Rats
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- Principles and standards for reporting animal experiments inThe Journal of PhysiologyandExperimental Physiology
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- Postsymptomatic restoration of SMN rescues the disease phenotype in a mouse model of severe spinal muscular atrophy
- (2011) Cathleen M. Lutz et al. JOURNAL OF CLINICAL INVESTIGATION
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- (2011) L. Liu-Yesucevitz et al. JOURNAL OF NEUROSCIENCE
- Sprouting capacity of lumbar motoneurons in normal and hemisected spinal cords of the rat
- (2010) T. Gordon et al. JOURNAL OF PHYSIOLOGY-LONDON
- Motoneuron survival after chronic and sequential peripheral nerve injuries in the rat
- (2009) Qing-Gui Xu et al. JOURNAL OF NEUROSURGERY
- Spinal muscular atrophy: why do low levels of survival motor neuron protein make motor neurons sick?
- (2009) Arthur H. M. Burghes et al. NATURE REVIEWS NEUROSCIENCE
- Functional over-load saves motor units in the SOD1-G93A transgenic mouse model of amyotrophic lateral sclerosis
- (2009) T. Gordon et al. NEUROBIOLOGY OF DISEASE
- SMN Deficiency Causes Tissue-Specific Perturbations in the Repertoire of snRNAs and Widespread Defects in Splicing
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- (2008) Shingo Kariya et al. HUMAN MOLECULAR GENETICS
- Exercise-Induced Activation of NMDA Receptor Promotes Motor Unit Development and Survival in a Type 2 Spinal Muscular Atrophy Model Mouse
- (2008) O. Biondi et al. JOURNAL OF NEUROSCIENCE
- Preferential motor unit loss in the SOD1G93Atransgenic mouse model of amyotrophic lateral sclerosis
- (2008) J. Hegedus et al. JOURNAL OF PHYSIOLOGY-LONDON
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- (2008) Mitchell R Lunn et al. LANCET
- The SMN binding protein gemin2 is not involved in motor axon outgrowth
- (2007) Michelle L. McWhorter et al. Developmental Neurobiology
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