Hypothalamus Specific Re-Introduction of SNORD116 into Otherwise Snord116 Deficient Mice Increased Energy Expenditure

Ambient temperature modulates the effects of the Prader-Willi syndrome candidate gene Snord116 on energy homeostasis

(2017) Y. Qi et al.   NEUROPEPTIDES

Non-coding RNAs and disease: the classical ncRNAs make a comeback

(2016) R. A. de Almeida et al.   BIOCHEMICAL SOCIETY TRANSACTIONS

Prader-Willi Critical Region, a Non-Translated, Imprinted Central Regulator of Bone Mass: Possible Role in Skeletal Abnormalities in Prader-Willi Syndrome

(2016) Ee-Cheng Khor et al.   PLoS One

Snord116 is critical in the regulation of food intake and body weight

(2016) Yue Qi et al.   Scientific Reports

SNORD116 and SNORD115 change expression of multiple genes and modify each other's activity

(2015) Marina Falaleeva et al.   GENE

Behavioural characteristics of the Prader–Willi syndrome related biallelic Snord116 mouse model

(2015) Jerzy Zieba et al.   NEUROPEPTIDES

Rapid Birth-and-Death Evolution of Imprinted snoRNAs in the Prader-Willi Syndrome Locus: Implications for Neural Development in Euarchontoglires

(2014) Yi-Jun Zhang et al.   PLoS One

Mouse models of Prader–Willi Syndrome: A systematic review

(2013) Sandrina Bervini et al.   FRONTIERS IN NEUROENDOCRINOLOGY

Hypothalamic expression of snoRNA Snord116 is consistent with a link to the hyperphagia and obesity symptoms of Prader–Willi syndrome

(2012) Qian Zhang et al.   INTERNATIONAL JOURNAL OF DEVELOPMENTAL NEUROSCIENCE

Unique and atypical deletions in Prader–Willi syndrome reveal distinct phenotypes

(2011) Soo-Jeong Kim et al.   EUROPEAN JOURNAL OF HUMAN GENETICS

Paternally inherited microdeletion at 15q11.2 confirms a significant role for the SNORD116 C/D box snoRNA cluster in Prader–Willi syndrome

(2010) Angela L Duker et al.   EUROPEAN JOURNAL OF HUMAN GENETICS

SnoRNA Snord116 (Pwcr1/MBII-85) Deletion Causes Growth Deficiency and Hyperphagia in Mice

(2008) Feng Ding et al.   PLoS One