Small Interfering RNA Targeting Dickkopf-1 Contributes to Neuroprotection After Intracerebral Hemorrhage in Rats

Matrigel and Activin A promote cell-cell contact and anti-apoptotic activity in cultured human retinal pigment epithelium cells

(2016) Xiaoling Guo et al.   EXPERIMENTAL EYE RESEARCH

Cytokine-mediated dysregulation of zonula occludens-1 properties in human brain microvascular endothelium

(2015) Keith D. Rochfort et al.   MICROVASCULAR RESEARCH

Agonists of epoxyeicosatrienoic acids reduce infarct size and ameliorate cardiac dysfunction via activation of HO-1 and Wnt1 canonical pathway

(2015) Jian Cao et al.   PROSTAGLANDINS & OTHER LIPID MEDIATORS

Promoting neurogenesis via Wnt/β-catenin signaling pathway accounts for the neurorestorative effects of morroniside against cerebral ischemia injury

(2014) Fang-Ling Sun et al.   EUROPEAN JOURNAL OF PHARMACOLOGY

Circulating Dickkopf‐1 in Diabetes Mellitus: Association With Platelet Activation and Effects of Improved Metabolic Control and Low‐Dose Aspirin

(2014) Stefano Lattanzio et al.   Journal of the American Heart Association

Loss of Dickkopf-1 Restores Neurogenesis in Old Age and Counteracts Cognitive Decline

(2013) Désirée R.M. Seib et al.   Cell Stem Cell

The Wnt/Planar Cell Polarity Signaling Pathway Contributes to the Integrity of Tight Junctions in Brain Endothelial Cells

(2013) Cédric Artus et al.   JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM

Premature myocardial infarction is associated with low serum levels of Wnt-1

(2012) Georg Goliasch et al.   ATHEROSCLEROSIS

Estrogen regulation of Dkk1 and Wnt/β-Catenin signaling in neurodegenerative disease

(2012) Erin L. Scott et al.   BRAIN RESEARCH

Constitutive overexpression of Norrin activates Wnt/β-catenin and endothelin-2 signaling to protect photoreceptors from light damage

(2012) Barbara M. Braunger et al.   NEUROBIOLOGY OF DISEASE

Wnt Signaling Enhances Neurogenesis and Improves Neurological Function after Focal Ischemic Injury

(2012) Adi Shruster et al.   PLoS One

Circulating Dickkopf-1 in acute ischemic stroke and clinically stable cerebrovascular disease

(2011) Thomas Seifert-Held et al.   ATHEROSCLEROSIS

Neutrophil Depletion Reduces Blood-Brain Barrier Breakdown, Axon Injury, and Inflammation After Intracerebral Hemorrhage

(2011) Iska Moxon-Emre et al.   JOURNAL OF NEUROPATHOLOGY AND EXPERIMENTAL NEUROLOGY

Increased serum Dickkopf-1 levels in drug-abusing psychotic patients

(2011) Daniele Serata et al.   PROGRESS IN NEURO-PSYCHOPHARMACOLOGY & BIOLOGICAL PSYCHIATRY

Inhibition of Carbonic Anhydrase Reduces Brain Injury After Intracerebral Hemorrhage

(2011) Fuyou Guo et al.   Translational Stroke Research

Looking at the blood–brain barrier: Molecular anatomy and possible investigation approaches

(2010) Filipa Lourenço Cardoso et al.   BRAIN RESEARCH REVIEWS

Mast Cells as Early Responders in the Regulation of Acute Blood–Brain Barrier Changes after Cerebral Ischemia and Hemorrhage

(2010) Perttu Johannes Lindsberg et al.   JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM

mRNA turnover rate limits siRNA and microRNA efficacy

(2010) Erik Larsson et al.   Molecular Systems Biology

Increased Dkk3 protein expression in platelets and megakaryocytes of patients with myeloproliferative neoplasms

(2010) M. Medinger et al.   THROMBOSIS AND HAEMOSTASIS

Dickkopf-1 Enhances Inflammatory Interaction Between Platelets and Endothelial Cells and Shows Increased Expression in Atherosclerosis

(2009) Thor Ueland et al.   ARTERIOSCLEROSIS THROMBOSIS AND VASCULAR BIOLOGY

Platelet is a major contributor to circulating levels of Dickkopf-1: clinical implications in patients with multiple myeloma

(2009) Nathalie Voorzanger-Rousselot et al.   BRITISH JOURNAL OF HAEMATOLOGY

Wnt/β-catenin signaling controls development of the blood–brain barrier

(2008) Stefan Liebner et al.   JOURNAL OF CELL BIOLOGY

Induction of the Wnt Antagonist, Dickkopf-1, Contributes to the Development of Neuronal Death in Models of Brain Focal Ischemia

(2008) Federica Mastroiacovo et al.   JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM