Journal
JOURNAL OF CELL BIOLOGY
Volume 216, Issue 11, Pages 3429-3431Publisher
ROCKEFELLER UNIV PRESS
DOI: 10.1083/jcb.201710068
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Funding
- Wellcome Trust [101200/Z/13/Z]
- Royal Society [101200/Z/13/Z]
- European Research Council [678350]
- Wellcome Trust [101200/Z/13/Z] Funding Source: Wellcome Trust
- European Research Council (ERC) [678350] Funding Source: European Research Council (ERC)
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Cancer cells thrive on genetic and epigenetic changes that confer a selective advantage but also need strategies to avoid immune recognition. In this issue, Cuellar et al. (2017. J. Cell Biol. https://doi.org/10.1083/jcb.201612160) find that the histone methyltransferase SET DB1 enables acute myeloid leukemia cells to evade sensing of retrotransposons by innate immune receptors.
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