CDX2 Stimulates the Proliferation of Porcine Intestinal Epithelial Cells by Activating the mTORC1 and Wnt/β-Catenin Signaling Pathways
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Title
CDX2 Stimulates the Proliferation of Porcine Intestinal Epithelial Cells by Activating the mTORC1 and Wnt/β-Catenin Signaling Pathways
Authors
Keywords
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Journal
INTERNATIONAL JOURNAL OF MOLECULAR SCIENCES
Volume 18, Issue 11, Pages 2447
Publisher
MDPI AG
Online
2017-11-21
DOI
10.3390/ijms18112447
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Note: Only part of the references are listed.- Activation of Wnt/β-catenin signalling is required for TGF-β/Smad2/3 signalling during myofibroblast proliferation
- (2017) Liang Xu et al. JOURNAL OF CELLULAR AND MOLECULAR MEDICINE
- L-Glutamate deficiency can trigger proliferation inhibition via down regulation of the mTOR/S6K1 pathway in pig intestinal epithelial cells1
- (2016) X.-G. Li et al. JOURNAL OF ANIMAL SCIENCE
- EAAT3 promotes amino acid transport and proliferation of porcine intestinal epithelial cells
- (2016) Jin-ling Ye et al. Oncotarget
- CDX2 increases SLC7A7 expression and proliferation of pig intestinal epithelial cells
- (2016) Xiang-guang Li et al. Oncotarget
- Porcine milk-derived exosomes promote proliferation of intestinal epithelial cells
- (2016) Ting Chen et al. Scientific Reports
- CDX2 inhibits pancreatic adenocarcinoma cell proliferation via promoting tumor suppressor miR-615-5p
- (2015) Yuanyuan Jiang et al. TUMOR BIOLOGY
- Distinct Processes and Transcriptional Targets Underlie CDX2 Requirements in Intestinal Stem Cells and Differentiated Villus Cells
- (2015) Adrianna K. San Roman et al. Stem Cell Reports
- Transformation of intestinal stem cells into gastric stem cells on loss of transcription factor Cdx2
- (2014) Salvatore Simmini et al. Nature Communications
- BAI3, CDX2 and VIL1: a panel of three antibodies to distinguish small cell from large cell neuroendocrine lung carcinomas
- (2013) Muhammad F Bari et al. HISTOPATHOLOGY
- PTEN expression and suppression of proliferation are associated with Cdx2 overexpression in gastric cancer cells
- (2013) ZHI-GANG BAI et al. INTERNATIONAL JOURNAL OF ONCOLOGY
- Association between vitamin D receptor gene Cdx2 polymorphism and breast cancer susceptibility
- (2013) Zhu-Chao Zhou et al. TUMOR BIOLOGY
- Cdx2 determines the fate of postnatal intestinal endoderm
- (2011) E. J. Stringer et al. DEVELOPMENT
- Mammalian target of rapamycin regulates expression of β-catenin in hepatocellular carcinoma
- (2011) Zhenbo Feng et al. HUMAN PATHOLOGY
- Intestine-specific transcription factor Cdx2 induces E-cadherin function by enhancing the trafficking of E-cadherin to the cell membrane
- (2010) Shinsuke Funakoshi et al. AMERICAN JOURNAL OF PHYSIOLOGY-GASTROINTESTINAL AND LIVER PHYSIOLOGY
- Cooperation between HNF-1α, Cdx2, and GATA-4 in initiating an enterocytic differentiation program in a normal human intestinal epithelial progenitor cell line
- (2010) Yannick D. Benoit et al. AMERICAN JOURNAL OF PHYSIOLOGY-GASTROINTESTINAL AND LIVER PHYSIOLOGY
- The role of CDX2 in intestinal homeostasis and inflammation
- (2010) Mehmet Coskun et al. BIOCHIMICA ET BIOPHYSICA ACTA-MOLECULAR BASIS OF DISEASE
- Genome-wide Analysis of CDX2 Binding in Intestinal Epithelial Cells (Caco-2)
- (2010) Mette Boyd et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- The intestine-specific transcription factor Cdx2 inhibits β-catenin/TCF transcriptional activity by disrupting the β-catenin–TCF protein complex
- (2009) Rong-Jun Guo et al. CARCINOGENESIS
- Cdx2 regulation of posterior development through non-Hox targets
- (2009) J. G. A. Savory et al. DEVELOPMENT
- Inhibition of the mTORC1 pathway suppresses intestinal polyp formation and reduces mortality in Apc 716 mice
- (2008) T. Fujishita et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
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