Anti-inflammatory mechanism of ulinastatin: Inhibiting the hyperpermeability of vascular endothelial cells induced by TNF-α via the RhoA/ROCK signal pathway
Anti-inflammatory mechanism of ulinastatin: Inhibiting the hyperpermeability of vascular endothelial cells induced by TNF-α via the RhoA/ROCK signal pathway
Authors
Keywords
Human umbilical vein endothelial cell line EA.hy926, High permeability, Ulinastatin, Tumor necrosis factor alpha (TNF-α), RhoA/ROCK signal pathway
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