Axonal plasticity underpins the functional recovery following surgical decompression in a rat model of cervical spondylotic myelopathy
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Title
Axonal plasticity underpins the functional recovery following surgical decompression in a rat model of cervical spondylotic myelopathy
Authors
Keywords
Cord Compression, Lesion Site, Cervical Spondylotic Myelopathy, Central Grey Matter, Compression Group
Journal
Acta Neuropathologica Communications
Volume 4, Issue 1, Pages -
Publisher
Springer Nature
Online
2016-08-23
DOI
10.1186/s40478-016-0359-7
References
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Note: Only part of the references are listed.- Does age affect surgical outcomes in patients with degenerative cervical myelopathy? Results from the prospective multicenter AOSpine International study on 479 patients
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- Riluzole blocks perioperative ischemia-reperfusion injury and enhances postdecompression outcomes in cervical spondylotic myelopathy
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- A Global Perspective on the Outcomes of Surgical Decompression in Patients With Cervical Spondylotic Myelopathy
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- The Prevalence and Phenotype of Activated Microglia/Macrophages within the Spinal Cord of the Hyperostotic Mouse (twy/twy) Changes in Response to Chronic Progressive Spinal Cord Compression: Implications for Human Cervical Compressive Myelopathy
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- Astrocytes regulate myelin clearance through recruitment of microglia during cuprizone-induced demyelination
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- Predictors of Surgical Outcome in Cervical Spondylotic Myelopathy
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- Human neuropathological and animal model evidence supporting a role for Fas-mediated apoptosis and inflammation in cervical spondylotic myelopathy
- (2011) W. R. Yu et al. BRAIN
- Synaptophysin Is Required for Synaptobrevin Retrieval during Synaptic Vesicle Endocytosis
- (2011) S. L. Gordon et al. JOURNAL OF NEUROSCIENCE
- Development and Characterization of a Novel Rat Model of Cervical Spondylotic Myelopathy: The Impact of Chronic Cord Compression on Clinical, Neuroanatomical, and Neurophysiological Outcomes
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- Reactive Astrocytes Form Scar-Like Perivascular Barriers to Leukocytes during Adaptive Immune Inflammation of the CNS
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