Loss of the oncogenic phosphatase PRL-3 promotes a TNF-R1 feedback loop that mediates triple-negative breast cancer growth
Published 2016 View Full Article
- Home
- Publications
- Publication Search
- Publication Details
Title
Loss of the oncogenic phosphatase PRL-3 promotes a TNF-R1 feedback loop that mediates triple-negative breast cancer growth
Authors
Keywords
-
Journal
Oncogenesis
Volume 5, Issue 8, Pages e255-e255
Publisher
Springer Nature
Online
2016-08-15
DOI
10.1038/oncsis.2016.50
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- Phosphatase PTP4A3 Promotes Triple-Negative Breast Cancer Growth and Predicts Poor Patient Survival
- (2016) Petra den Hollander et al. CANCER RESEARCH
- Reciprocal leukemia-stroma VCAM-1/VLA-4-dependent activation of NF- B mediates chemoresistance
- (2014) R. Jacamo et al. BLOOD
- The value of platinum agents as neoadjuvant chemotherapy in triple-negative breast cancers: a systematic review and meta-analysis
- (2014) Fausto Petrelli et al. BREAST CANCER RESEARCH AND TREATMENT
- Platinum-based chemotherapy in triple-negative advanced breast cancer
- (2014) Cynthia Villarreal-Garza et al. BREAST CANCER RESEARCH AND TREATMENT
- Reduced expression of GDF-15 is associated with atrophic inflammatory lesions of the prostate
- (2014) James R. Lambert et al. PROSTATE
- AP-1/c-Jun transcription factors: Regulation and function in malignant melanoma
- (2013) Melanie Kappelmann et al. EUROPEAN JOURNAL OF CELL BIOLOGY
- PRL-3 activates NF-κB signaling pathway by interacting with RAP1
- (2012) Shenyi Lian et al. BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS
- SnapShot: Breast Cancer
- (2012) Kornelia Polyak et al. CANCER CELL
- Comprehensive molecular portraits of human breast tumours
- (2012) Daniel C. Koboldt et al. NATURE
- 1α,25-Dihydroxyvitamin D3-3β-bromoacetate, a potential cancer therapeutic agent: Synthesis and molecular mechanism of action
- (2011) Rahul Ray et al. BIOORGANIC & MEDICINAL CHEMISTRY LETTERS
- The Two Faces of NF-κB Signaling in Cancer Development and Therapy
- (2011) Ulf Klein et al. CANCER CELL
- Emerging role of NF-κB signaling in the induction of senescence-associated secretory phenotype (SASP)
- (2011) Antero Salminen et al. CELLULAR SIGNALLING
- Control of the senescence-associated secretory phenotype by NF- B promotes senescence and enhances chemosensitivity
- (2011) Y. Chien et al. GENES & DEVELOPMENT
- Treatment of triple-negative metastatic breast cancer: toward individualized targeted treatments or chemosensitization?
- (2010) N. Berrada et al. ANNALS OF ONCOLOGY
- The essence of senescence
- (2010) T. Kuilman et al. GENES & DEVELOPMENT
- RelA/p65 functions to maintain cellular senescence by regulating genomic stability and DNA repair
- (2009) Jingxin Wang et al. EMBO REPORTS
- A PTP4A3 Peptide PIMAP39 Modulates TNF-Alpha Levels and Endotoxic Shock
- (2009) Xiaoren Tang et al. Journal of Innate Immunity
- The Metastasis-Associated Gene Prl-3 Is a p53 Target Involved in Cell-Cycle Regulation
- (2008) Shashwati Basak et al. MOLECULAR CELL
- Senescence-Associated Secretory Phenotypes Reveal Cell-Nonautonomous Functions of Oncogenic RAS and the p53 Tumor Suppressor
- (2008) Jean-Philippe Coppé et al. PLOS BIOLOGY
Find Funding. Review Successful Grants.
Explore over 25,000 new funding opportunities and over 6,000,000 successful grants.
ExploreFind the ideal target journal for your manuscript
Explore over 38,000 international journals covering a vast array of academic fields.
Search