Uric Acid-Induced Adipocyte Dysfunction Is Attenuated by HO-1 Upregulation: Potential Role of Antioxidant Therapy to Target Obesity
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Title
Uric Acid-Induced Adipocyte Dysfunction Is Attenuated by HO-1 Upregulation: Potential Role of Antioxidant Therapy to Target Obesity
Authors
Keywords
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Journal
Stem Cells International
Volume 2016, Issue -, Pages 1-11
Publisher
Hindawi Limited
Online
2015-11-23
DOI
10.1155/2016/8197325
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Related references
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- (2012) T. Ishimoto et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
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- Oxidative stress and diabetic cardiovascular disorders: roles of mitochondria and NADPH oxidaseThis review is one of a selection of papers published in a Special Issue on Oxidative Stress in Health and Disease.
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- HO-1 expression increases mesenchymal stem cell-derived osteoblasts but decreases adipocyte lineage
- (2009) Luca Vanella et al. BONE
- Fructose consumption: potential mechanisms for its effects to increase visceral adiposity and induce dyslipidemia and insulin resistance
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- Wnt/β-Catenin Signaling: Components, Mechanisms, and Diseases
- (2009) Bryan T. MacDonald et al. DEVELOPMENTAL CELL
- Hypothesis: Could Excessive Fructose Intake and Uric Acid Cause Type 2 Diabetes?
- (2009) Richard J. Johnson et al. ENDOCRINE REVIEWS
- Recent insights into the pathogenesis of hyperuricaemia and gout
- (2009) P. L. Riches et al. HUMAN MOLECULAR GENETICS
- Heme Oxygenase-1 Induction Remodels Adipose Tissue and Improves Insulin Sensitivity in Obesity-Induced Diabetic Rats
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- (2009) Stephen J. Peterson et al. JOURNAL OF LIPID RESEARCH
- Carbon Monoxide Blocks Lipopolysaccharide-Induced Gene Expression by Interfering with Proximal TLR4 to NF-κB Signal Transduction in Human Monocytes
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- Treatment of Obese Diabetic Mice With a Heme Oxygenase Inducer Reduces Visceral and Subcutaneous Adiposity, Increases Adiponectin Levels, and Improves Insulin Sensitivity and Glucose Tolerance
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- Uric Acid: The Oxidant-Antioxidant Paradox
- (2008) Yuri Y. Sautin et al. NUCLEOSIDES NUCLEOTIDES & NUCLEIC ACIDS
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