Deubiquitinase MYSM1 Is Essential for Normal Bone Formation and Mesenchymal Stem Cell Differentiation
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Title
Deubiquitinase MYSM1 Is Essential for Normal Bone Formation and Mesenchymal Stem Cell Differentiation
Authors
Keywords
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Journal
Scientific Reports
Volume 6, Issue 1, Pages -
Publisher
Springer Nature
Online
2016-02-26
DOI
10.1038/srep22211
References
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Related references
Note: Only part of the references are listed.- Repression of p53-target gene Bbc3/PUMA by MYSM1 is essential for the survival of hematopoietic multipotent progenitors and contributes to stem cell maintenance
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- Deubiquitinase MYSM1 Regulates Innate Immunity through Inactivation of TRAF3 and TRAF6 Complexes
- (2015) Swarupa Panda et al. IMMUNITY
- An in vivo genetic reversion highlights the crucial role of Myb-Like, SWIRM, and MPN domains 1 (MYSM1) in human hematopoiesis and lymphocyte differentiation
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- S-nitrosoglutathione reductase–dependent PPARγ denitrosylation participates in MSC-derived adipogenesis and osteogenesis
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- p53 Loss Increases the Osteogenic Differentiation of Bone Marrow Stromal Cells
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- MYSM1 is mutated in a family with transient transfusion-dependent anemia, mild thrombocytopenia, and low NK- and B-cell counts
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- (2010) A. Michael Parfitt et al. JOURNAL OF BONE AND MINERAL RESEARCH
- Histone H3K27 methyltransferase Ezh2 represses Wnt genes to facilitate adipogenesis
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