Melittin, the Major Pain-Producing Substance of Bee Venom

Activation of Tetrodotoxin-Resistant Sodium Channel NaV1.9 in Rat Primary Sensory Neurons Contributes to Melittin-Induced Pain Behavior

(2012) Yao-Qing Yu et al.   NEUROMOLECULAR MEDICINE

Effects of a non-selective TRPC channel blocker, SKF-96365, on melittininduced spontaneous persistent nociception and inflammatory pain hypersensitivity

(2012) Jing Ding et al.   Neuroscience Bulletin

Melittin activates TRPV1 receptors in primary nociceptive sensory neurons via the phospholipase A2 cascade pathways

(2011) Yi-Ru Du et al.   BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS

Antisense-Mediated Knockdown of NaV1.8, but Not NaV1.9, Generates Inhibitory Effects on Complete Freund's Adjuvant-Induced Inflammatory Pain in Rat

(2011) Yao-Qing Yu et al.   PLoS One

Effects of SKF-96365, a TRPC inhibitor, on melittin-induced inward current and intracellular Ca2+ rise in primary sensory cells

(2011) Jing Ding et al.   Neuroscience Bulletin

The nociceptive and anti-nociceptive effects of bee venom injection and therapy: A double-edged sword

(2010) Jun Chen et al.   PROGRESS IN NEUROBIOLOGY

Thermodynamics of Melittin Binding to Lipid Bilayers. Aggregation and Pore Formation†

(2009) Gabriela Klocek et al.   BIOCHEMISTRY

Activation of ERK1/2 in the primary injury site is required to maintain melittin-enhanced wind-up of rat spinal wide-dynamic-range neurons

(2009) Yao-Qing Yu et al.   NEUROSCIENCE LETTERS

Extracellular signal-regulated kinases mediate melittin-induced hypersensitivity of spinal neurons to chemical and thermal but not mechanical stimuli

(2008) Meng-Meng Li et al.   BRAIN RESEARCH BULLETIN

Roles of Peripheral P2X and P2Y Receptors in the Development of Melittin-Induced Nociception and Hypersensitivity

(2008) Zhuo-Min Lu et al.   NEUROCHEMICAL RESEARCH

Roles of peripheral mitogen-activated protein kinases in melittin-induced nociception and hyperalgesia

(2008) J. Hao et al.   NEUROSCIENCE