CD169+ macrophages regulate PD-L1 expression via type I interferon and thereby prevent severe immunopathology after LCMV infection

CEACAM1 induces B-cell survival and is essential for protective antiviral antibody production

(2015) Vishal Khairnar et al.   Nature Communications

PD-L1 Expression on Retrovirus-Infected Cells Mediates Immune Escape from CD8+ T Cell Killing

(2015) Ilseyar Akhmetzyanova et al.   PLoS Pathogens

Type I Interferon Protects Antiviral CD8+ T Cells from NK Cell Cytotoxicity

(2014) Haifeng C. Xu et al.   IMMUNITY

Blockade of Chronic Type I Interferon Signaling to Control Persistent LCMV Infection

(2013) E. B. Wilson et al.   SCIENCE

Persistent LCMV Infection Is Controlled by Blockade of Type I Interferon Signaling

(2013) J. R. Teijaro et al.   SCIENCE

Usp18 Driven Enforced Viral Replication in Dendritic Cells Contributes to Break of Immunological Tolerance in Autoimmune Diabetes

(2013) Nadine Honke et al.   PLoS Pathogens

Tunicamycin Inhibits Diabetes

(2012) Namir Shaabani et al.   CELLULAR PHYSIOLOGY AND BIOCHEMISTRY

Virus-Specific CD8+ T Cells Upregulate Programmed Death-1 Expression during Acute Friend Retrovirus Infection but Are Highly Cytotoxic and Control Virus Replication

(2011) G. Zelinskyy et al.   JOURNAL OF IMMUNOLOGY

Enforced viral replication activates adaptive immunity and is essential for the control of a cytopathic virus

(2011) Nadine Honke et al.   NATURE IMMUNOLOGY

Tissue macrophages suppress viral replication and prevent severe immunopathology in an interferon-I-dependent manner in mice

(2010) Philipp A. Lang et al.   HEPATOLOGY

Visualization of IFN  production by plasmacytoid versus conventional dendritic cells under specific stimulation conditions in vivo

(2008) S. Scheu et al.   PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA