Fecal Lipocalin-2 as a Sensitive and Noninvasive Biomarker in the TNBS Crohn’s Inflammatory Bowel Disease Model

Inflammatory Bowel Disease: Genetics, Epigenetics, and Pathogenesis

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Diagnosis and classification of Crohn's disease

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Sa1216 Faecal Lipocalin-2 As Biomarker in the Evaluation of Crohn's Disease Activity

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Relationship Between Neutrophil Gelatinase-Associated Lipocalin (NGAL) Levels and Inflammatory Bowel Disease Type and Activity

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Early detection of renal injury using urinary vanin-1 in rats with experimental colitis

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Murine Models of Inflammatory Bowel Disease (IBD)

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Biomarkers of Therapeutic Response in the IL-23 Pathway in Inflammatory Bowel Disease

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The biodiversity and composition of the dominant fecal microbiota in patients with inflammatory bowel disease

(2012) Sumei Sha et al.   DIAGNOSTIC MICROBIOLOGY AND INFECTIOUS DISEASE

Terminal Restriction Fragment Length Polymorphism Analysis of the Gut Microbiota Profiles of Pediatric Patients with Inflammatory Bowel Disease

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Recent Trends in the Prevalence of Crohn’s Disease and Ulcerative Colitis in a Commercially Insured US Population

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Fecal Lipocalin 2, a Sensitive and Broadly Dynamic Non-Invasive Biomarker for Intestinal Inflammation

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High-throughput clone library analysis of the mucosa-associated microbiota reveals dysbiosis and differences between inflamed and non-inflamed regions of the intestine in inflammatory bowel disease

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Pharmacologic Evaluation of Sulfasalazine, FTY720, and Anti-IL-12/23p40 in a TNBS-Induced Crohn’s Disease Model

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Neutrophil gelatinase-associated lipocalin (NGAL) in inflammatory bowel disease: association with pathophysiology of inflammation, established markers, and disease activity

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Association Between the Use of Antibiotics in the First Year of Life and Pediatric Inflammatory Bowel Disease

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Antibiotic use and inflammatory bowel diseases in childhood

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Accuracy of Neutrophil Gelatinase-Associated Lipocalin (NGAL) in Diagnosis and Prognosis in Acute Kidney Injury: A Systematic Review and Meta-analysis

(2009) Michael Haase et al.   AMERICAN JOURNAL OF KIDNEY DISEASES

Deletion polymorphism upstream of IRGM associated with altered IRGM expression and Crohn's disease

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