Defective trained immunity in patients with STAT-1-dependent chronic mucocutaneaous candidiasis

BCG-induced trained immunity in NK cells: Role for non-specific protection to infection

(2014) Johanneke Kleinnijenhuis et al.   CLINICAL IMMUNOLOGY

Innate immune memory: towards a better understanding of host defense mechanisms

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A novel large deletion of the DOCK8 gene in a Chinese family with autosomal-recessive hyper-IgE syndrome

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mTOR- and HIF-1 -mediated aerobic glycolysis as metabolic basis for trained immunity

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Epigenetic programming of monocyte-to-macrophage differentiation and trained innate immunity

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New and recurrent gain-of-functionSTAT1mutations in patients with chronic mucocutaneous candidiasis from Eastern and Central Europe

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(2012) Kathryn J. Sowerwine et al.   Annals of the New York Academy of Sciences

Candida albicans Infection Affords Protection against Reinfection via Functional Reprogramming of Monocytes

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Trained Immunity: A Memory for Innate Host Defense

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Gain-of-function humanSTAT1mutations impair IL-17 immunity and underlie chronic mucocutaneous candidiasis

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STAT1Mutations in Autosomal Dominant Chronic Mucocutaneous Candidiasis

(2011) Frank L. van de Veerdonk et al.   NEW ENGLAND JOURNAL OF MEDICINE

IFN-  abrogates endotoxin tolerance by facilitating Toll-like receptor-induced chromatin remodeling

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Menin and RNF20 recruitment is associated with dynamic histone modifications that regulate signal transducer and activator of transcription 1 (STAT1)-activated transcription of the interferon regulatory factor 1 gene (IRF1)

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Th17 cells and IL-17 receptor signaling are essential for mucosal host defense against oral candidiasis

(2009) Heather R. Conti et al.   JOURNAL OF EXPERIMENTAL MEDICINE

Clinical Manifestations, Etiology, and Pathogenesis of the Hyper-IgE Syndromes

(2009) Alexandra F Freeman et al.   PEDIATRIC RESEARCH

Hyper IgE syndrome: an update on clinical aspects and the role of signal transducer and activator of transcription 3

(2008) Michelle L Paulson et al.   Current Opinion in Allergy and Clinical Immunology

Deficiency of Th17 cells in hyper IgE syndrome due to mutations inSTAT3

(2008) Cindy S. Ma et al.   JOURNAL OF EXPERIMENTAL MEDICINE

Mutations inSTAT3andIL12RB1impair the development of human IL-17–producing T cells

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Impaired TH17 cell differentiation in subjects with autosomal dominant hyper-IgE syndrome

(2008) Joshua D. Milner et al.   NATURE