ST8SIA4-Dependent Polysialylation is Part of a Developmental Program Required for Germ Layer Formation from Human Pluripotent Stem Cells
Published 2016 View Full Article
- Home
- Publications
- Publication Search
- Publication Details
Title
ST8SIA4-Dependent Polysialylation is Part of a Developmental Program Required for Germ Layer Formation from Human Pluripotent Stem Cells
Authors
Keywords
-
Journal
STEM CELLS
Volume 34, Issue 7, Pages 1742-1752
Publisher
Wiley
Online
2016-04-13
DOI
10.1002/stem.2379
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- Evidences for the involvement of cell surface glycans in stem cell pluripotency and differentiation
- (2014) F. Alisson-Silva et al. GLYCOBIOLOGY
- ST6Gal-I Protein Expression Is Upregulated in Human Epithelial Tumors and Correlates with Stem Cell Markers in Normal Tissues and Colon Cancer Cell Lines
- (2013) A. F. Swindall et al. CANCER RESEARCH
- Genome engineering using the CRISPR-Cas9 system
- (2013) F Ann Ran et al. Nature Protocols
- Polysialic Acid: Versatile Modification of NCAM, SynCAM 1 and Neuropilin-2
- (2013) Martina Mühlenhoff et al. NEUROCHEMICAL RESEARCH
- RNA-Guided Human Genome Engineering via Cas9
- (2013) P. Mali et al. SCIENCE
- ENCODE Data in the UCSC Genome Browser: year 5 update
- (2012) Kate R. Rosenbloom et al. NUCLEIC ACIDS RESEARCH
- Specific lectin biomarkers for isolation of human pluripotent stem cells identified through array-based glycomic analysis
- (2011) Yu-Chieh Wang et al. CELL RESEARCH
- Polysialylation promotes neural cell adhesion molecule-mediated cell migration in a fibroblast growth factor receptor-dependent manner, but independent of adhesion capability
- (2011) J. Li et al. GLYCOBIOLOGY
- Glycome Diagnosis of Human Induced Pluripotent Stem Cells Using Lectin Microarray
- (2011) Hiroaki Tateno et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Novel Regulation of Fibroblast Growth Factor 2 (FGF2)-mediated Cell Growth by Polysialic Acid
- (2011) Sayaka Ono et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Polysialic acid controls NCAM signals at cell-cell contacts to regulate focal adhesion independent from FGF receptor activity
- (2011) K. Eggers et al. JOURNAL OF CELL SCIENCE
- Wnt signaling and a Smad pathway blockade direct the differentiation of human pluripotent stem cells to multipotent neural crest cells
- (2011) L. Menendez et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Polysialic Acid Neural Cell Adhesion Molecule (PSA-NCAM) is an adverse prognosis factor in glioblastoma, and regulates olig2 expression in glioma cell lines
- (2010) Marie-Claude Amoureux et al. BMC CANCER
- The T-box transcription factor Brachyury promotes epithelial-mesenchymal transition in human tumor cells
- (2010) Romaine I. Fernando et al. JOURNAL OF CLINICAL INVESTIGATION
- Synaptic cell adhesion molecule SynCAM 1 is a target for polysialylation in postnatal mouse brain
- (2010) S. P. Galuska et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Highly efficient neural conversion of human ES and iPS cells by dual inhibition of SMAD signaling
- (2009) Stuart M Chambers et al. NATURE BIOTECHNOLOGY
- NCAM-induced focal adhesion assembly: a functional switch upon loss of E-cadherin
- (2008) Francois Lehembre et al. EMBO JOURNAL
- Polysialylated NCAM Represses E-Cadherin-Mediated Cell-Cell Adhesion in Pancreatic Tumor Cells
- (2008) Susanne C. Schreiber et al. GASTROENTEROLOGY
- Null mutations in human and mouse orthologs frequently result in different phenotypes
- (2008) B.-Y. Liao et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Polysialic acid in the plasticity of the developing and adult vertebrate nervous system
- (2007) Urs Rutishauser NATURE REVIEWS NEUROSCIENCE
Discover Peeref hubs
Discuss science. Find collaborators. Network.
Join a conversationCreate your own webinar
Interested in hosting your own webinar? Check the schedule and propose your idea to the Peeref Content Team.
Create Now