Redox dysregulation, neuroinflammation, and NMDA receptor hypofunction: A “central hub” in schizophrenia pathophysiology?

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Title
Redox dysregulation, neuroinflammation, and NMDA receptor hypofunction: A “central hub” in schizophrenia pathophysiology?
Authors
Keywords
Parvalbumin interneurons, Oligodendrocytes, Myelination, N-acetylcysteine, Development, Oxidative stress
Journal
SCHIZOPHRENIA RESEARCH
Volume 176, Issue 1, Pages 41-51
Publisher
Elsevier BV
Online
2014-07-05
DOI
10.1016/j.schres.2014.06.021

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