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Title
Characterization of genetic loss-of-function of Fus in zebrafish
Authors
Keywords
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Journal
RNA Biology
Volume 14, Issue 1, Pages 29-35
Publisher
Informa UK Limited
Online
2016-11-30
DOI
10.1080/15476286.2016.1256532
References
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- (2015) Akio Masuda et al. GENES & DEVELOPMENT
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- (2014) Jürgen Cox et al. MOLECULAR & CELLULAR PROTEOMICS
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- featureCounts: an efficient general purpose program for assigning sequence reads to genomic features
- (2013) Y. Liao et al. BIOINFORMATICS
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- (2013) Gary A.B. Armstrong et al. HUMAN MOLECULAR GENETICS
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- (2013) Woong Y Hwang et al. NATURE BIOTECHNOLOGY
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- (2009) C. Vance et al. SCIENCE
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