Sam68 Mediates the Activation of Insulin and Leptin Signalling in Breast Cancer Cells
Published 2016 View Full Article
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Title
Sam68 Mediates the Activation of Insulin and Leptin Signalling in Breast Cancer Cells
Authors
Keywords
Insulin, Leptin, Small interfering RNAs, Breast cancer, Insulin signaling, MAPK signaling cascades, Phosphorylation, Hormones
Journal
PLoS One
Volume 11, Issue 7, Pages e0158218
Publisher
Public Library of Science (PLoS)
Online
2016-07-15
DOI
10.1371/journal.pone.0158218
References
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Related references
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- (2013) Flora Sánchez-Jiménez et al. INTERNATIONAL JOURNAL OF MOLECULAR SCIENCES
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- The cellular and molecular mechanisms by which insulin influences breast cancer risk and progression
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- Down-regulation of RIP expression by 17-dimethylaminoethylamino-17-demethoxygeldanamycin promotes TRAIL-induced apoptosis in breast tumor cells
- (2009) Carmen Palacios et al. CANCER LETTERS
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- (2009) M. P. Paronetto et al. CANCER RESEARCH
- Sam68 regulates translation of target mRNAs in male germ cells, necessary for mouse spermatogenesis
- (2009) Maria Paola Paronetto et al. JOURNAL OF CELL BIOLOGY
- Leptin signaling in breast cancer: An overview
- (2008) Donatella Cirillo et al. JOURNAL OF CELLULAR BIOCHEMISTRY
- Sam68 Regulates a Set of Alternatively Spliced Exons during Neurogenesis
- (2008) G. Chawla et al. MOLECULAR AND CELLULAR BIOLOGY
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