Journal
PHYSIOLOGY
Volume 31, Issue 2, Pages 95-107Publisher
AMER PHYSIOLOGICAL SOC
DOI: 10.1152/physiol.00034.2015
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Funding
- United States Department of Veteran's Affairs Rehabilitation Research and Development Service [I01BX007080]
- VA Merit Award [E1075-R]
- National Institutes of Health [R01 AG-006457 29]
- Medical Research Foundation of Oregon
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People with Parkinson's disease exhibit debilitating gait impairments, including gait slowness, increased step variability, and poor postural control. A widespread supraspinal locomotor network including the cortex, cerebellum, basal ganglia, and brain stem contributes to the control of human locomotion, and altered activity of these structures underlies gait dysfunction due to Parkinson's disease.
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