Both the H 2 S biosynthesis inhibitor aminooxyacetic acid and the mitochondrially targeted H 2 S donor AP39 exert protective effects in a mouse model of burn injury

Published 2016 View Full Article

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Title
Both the H 2 S biosynthesis inhibitor aminooxyacetic acid and the mitochondrially targeted H 2 S donor AP39 exert protective effects in a mouse model of burn injury
Authors
Keywords
Hydrogen sulfide, Thermal injury, Shock, Inflammation, Nitric oxide
Journal
PHARMACOLOGICAL RESEARCH
Volume 113, Issue -, Pages 348-355
Publisher
Elsevier BV
Online
2016-09-16
DOI
10.1016/j.phrs.2016.09.013

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