Ctp1-dependent clipping and resection of DNA double-strand breaks by Mre11 endonuclease complex are not genetically separable
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Title
Ctp1-dependent clipping and resection of DNA double-strand breaks by Mre11 endonuclease complex are not genetically separable
Authors
Keywords
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Journal
NUCLEIC ACIDS RESEARCH
Volume 44, Issue 17, Pages 8241-8249
Publisher
Oxford University Press (OUP)
Online
2016-06-22
DOI
10.1093/nar/gkw557
References
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Related references
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- Sae2 promotes dsDNA endonuclease activity within Mre11–Rad50–Xrs2 to resect DNA breaks
- (2014) Elda Cannavo et al. NATURE
- Mechanism and Regulation of Meiotic Recombination Initiation
- (2014) Isabel Lam et al. Cold Spring Harbor Perspectives in Biology
- Double-Strand Break End Resection and Repair Pathway Choice
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- Regulatory networks integrating cell cycle control with DNA damage checkpoints and double-strand break repair
- (2011) P. Langerak et al. PHILOSOPHICAL TRANSACTIONS OF THE ROYAL SOCIETY B-BIOLOGICAL SCIENCES
- SUMO-Targeted Ubiquitin Ligase, Rad60, and Nse2 SUMO Ligase Suppress Spontaneous Top1–Mediated DNA Damage and Genome Instability
- (2011) Johanna Heideker et al. PLoS Genetics
- Release of Ku and MRN from DNA Ends by Mre11 Nuclease Activity and Ctp1 Is Required for Homologous Recombination Repair of Double-Strand Breaks
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- Regulation of Homologous Recombination in Eukaryotes
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- Ku prevents Exo1 and Sgs1-dependent resection of DNA ends in the absence of a functional MRX complex or Sae2
- (2010) Eleni P Mimitou et al. EMBO JOURNAL
- Nbs1 Flexibly Tethers Ctp1 and Mre11-Rad50 to Coordinate DNA Double-Strand Break Processing and Repair
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- Mre11 Dimers Coordinate DNA End Bridging and Nuclease Processing in Double-Strand-Break Repair
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