A defect in homologous recombination leads to increased translesion synthesisin E. coli

Single-strand gap repair involves both RecF and RecBCD pathways

(2016) Vincent Pagès   CURRENT GENETICS

Bacterial Proliferation: Keep Dividing and Don't Mind the Gap

(2015) Luisa Laureti et al.   PLoS Genetics

DNA polymerase IV mediates efficient and quick recovery of replication forks stalled at N2-dG adducts

(2014) Mio Ikeda et al.   NUCLEIC ACIDS RESEARCH

Chronology in lesion tolerance gives priority to genetic variability

(2014) K. Naiman et al.   PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA

Dynamics of Leading-Strand Lesion Skipping by the Replisome

(2013) Joseph T.P. Yeeles et al.   MOLECULAR CELL

Competition, collaboration and coordination - determining how cells bypass DNA damage

(2012) J. E. Sale   JOURNAL OF CELL SCIENCE

Monitoring bypass of single replication-blocking lesions by damage avoidance in the Escherichia coli chromosome

(2012) Vincent Pagès et al.   NUCLEIC ACIDS RESEARCH

Translational Control in Cancer Etiology

(2012) D. Ruggero   Cold Spring Harbor Perspectives in Biology

Separation of Recombination and SOS Response in Escherichia coli RecA Suggests LexA Interaction Sites

(2011) Anbu K. Adikesavan et al.   PLoS Genetics

The active form of DNA polymerase V is UmuD′2C–RecA–ATP

(2009) Qingfei Jiang et al.   NATURE

Biochemical basis for the essential genetic requirements of RecA and the  -clamp in Pol V activation

(2009) S. Fujii et al.   PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA