Adropin inhibits the progression of atherosclerosis in ApoE-/-/Enho-/- mice by regulating endothelial-to-mesenchymal transition
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Title
Adropin inhibits the progression of atherosclerosis in ApoE-/-/Enho-/- mice by regulating endothelial-to-mesenchymal transition
Authors
Keywords
-
Journal
Cell Death Discovery
Volume 9, Issue 1, Pages -
Publisher
Springer Science and Business Media LLC
Online
2023-10-31
DOI
10.1038/s41420-023-01697-3
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Note: Only part of the references are listed.- Adropin-based dual treatment enhances the therapeutic potential of mesenchymal stem cells in rat myocardial infarction
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- Endothelial to mesenchymal transition is common in atherosclerotic lesions and is associated with plaque instability
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- Endothelial-to-mesenchymal transition contributes to fibro-proliferative vascular disease and is modulated by fluid shear stress
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- (2014) S. Gao et al. DIABETES
- Adropin Is a Brain Membrane-bound Protein Regulating Physical Activity via the NB-3/Notch Signaling Pathway in Mice
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- TGF- Signaling Mediates Endothelial-to-Mesenchymal Transition (EndMT) During Vein Graft Remodeling
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- Adropin Deficiency Is Associated With Increased Adiposity and Insulin Resistance
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- (2008) K. Ganesh Kumar et al. Cell Metabolism
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