CD69 controls the uptake of L-tryptophan through LAT1-CD98 and AhR-dependent secretion of IL-22 in psoriasis
Published 2016 View Full Article
- Home
- Publications
- Publication Search
- Publication Details
Title
CD69 controls the uptake of L-tryptophan through LAT1-CD98 and AhR-dependent secretion of IL-22 in psoriasis
Authors
Keywords
-
Journal
NATURE IMMUNOLOGY
Volume 17, Issue 8, Pages 985-996
Publisher
Springer Nature
Online
2016-07-04
DOI
10.1038/ni.3504
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- The tryptophan metabolism enzyme L-kynureninase is a novel inflammatory factor in psoriasis and other inflammatory diseases
- (2016) Jamie L. Harden et al. JOURNAL OF ALLERGY AND CLINICAL IMMUNOLOGY
- Evidence for New Light-Independent Pathways for Generation of the Endogenous Aryl Hydrocarbon Receptor Agonist FICZ
- (2015) Anna Smirnova et al. CHEMICAL RESEARCH IN TOXICOLOGY
- Gene regulation of filaggrin and other skin barrier proteins via aryl hydrocarbon receptor
- (2015) Masutaka Furue et al. JOURNAL OF DERMATOLOGICAL SCIENCE
- Genetic and Pharmacological Analysis Identifies a Physiological Role for the AHR in Epidermal Differentiation
- (2015) Ellen H. van den Bogaard et al. JOURNAL OF INVESTIGATIVE DERMATOLOGY
- Metabolic control of type 1 regulatory T cell differentiation by AHR and HIF1-α
- (2015) Ivan D Mascanfroni et al. NATURE MEDICINE
- Immunology of Psoriasis
- (2014) Michelle A. Lowes et al. Annual Review of Immunology
- Activation of the Aryl Hydrocarbon Receptor Dampens the Severity of Inflammatory Skin Conditions
- (2014) Paola Di Meglio et al. IMMUNITY
- IL-22 Fate Reporter Reveals Origin and Control of IL-22 Production in Homeostasis and Infection
- (2014) Helena Ahlfors et al. JOURNAL OF IMMUNOLOGY
- Psoriasis
- (2014) P. Di Meglio et al. Cold Spring Harbor Perspectives in Medicine
- Role of amino acid transporters in amino acid sensing
- (2013) Peter M Taylor AMERICAN JOURNAL OF CLINICAL NUTRITION
- Copy number variations in IL22 gene are associated with Psoriasis vulgaris
- (2013) Ele Prans et al. HUMAN IMMUNOLOGY
- IL-22, not simply a Th17 cytokine
- (2013) Sascha Rutz et al. IMMUNOLOGICAL REVIEWS
- Control of amino-acid transport by antigen receptors coordinates the metabolic reprogramming essential for T cell differentiation
- (2013) Linda V Sinclair et al. NATURE IMMUNOLOGY
- The developmental pathway for CD103+CD8+ tissue-resident memory T cells of skin
- (2013) Laura K Mackay et al. NATURE IMMUNOLOGY
- Is CD69 an effective brake to control inflammatory diseases?
- (2013) Roberto González-Amaro et al. TRENDS IN MOLECULAR MEDICINE
- Aldara activates TLR7-independent immune defence
- (2013) Anne Walter et al. Nature Communications
- Molecular Mechanisms of Differentiation of Murine Pro-Inflammatory γδ T Cell Subsets
- (2013) Karine Serre et al. Frontiers in Immunology
- IL-22 induced cell proliferation is regulated by PI3K/Akt/mTOR signaling cascade
- (2012) Anupam Mitra et al. CYTOKINE
- Rorγt+ innate lymphocytes and γδ T cells initiate psoriasiform plaque formation in mice
- (2012) Stanislav Pantelyushin et al. JOURNAL OF CLINICAL INVESTIGATION
- PDK1 regulation of mTOR and hypoxia-inducible factor 1 integrate metabolism and migration of CD8+T cells
- (2012) David K. Finlay et al. JOURNAL OF EXPERIMENTAL MEDICINE
- CD69 Regulates Type I IFN-Induced Tolerogenic Signals to Mucosal CD4 T Cells That Attenuate Their Colitogenic Potential
- (2012) K. Radulovic et al. JOURNAL OF IMMUNOLOGY
- Anti–Interleukin-17 Monoclonal Antibody Ixekizumab in Chronic Plaque Psoriasis
- (2012) Craig Leonardi et al. NEW ENGLAND JOURNAL OF MEDICINE
- Notch-Hes1 pathway is required for the development of IL-17-producing T cells
- (2011) K. Shibata et al. BLOOD
- Pivotal Role of Dermal IL-17-Producing γδ T Cells in Skin Inflammation
- (2011) Yihua Cai et al. IMMUNITY
- The Aryl Hydrocarbon Receptor Regulates Gut Immunity through Modulation of Innate Lymphoid Cells
- (2011) Ju Qiu et al. IMMUNITY
- Cutaneous immunosurveillance by self-renewing dermal γδ T cells
- (2011) Nital Sumaria et al. JOURNAL OF EXPERIMENTAL MEDICINE
- IL-22 Is Required for Imiquimod-Induced Psoriasiform Skin Inflammation in Mice
- (2011) A. B. Van Belle et al. JOURNAL OF IMMUNOLOGY
- Identification of a Novel Proinflammatory Human Skin-Homing V 9V 2 T Cell Subset with a Potential Role in Psoriasis
- (2011) U. Laggner et al. JOURNAL OF IMMUNOLOGY
- CD69 Limits the Severity of Cardiomyopathy After Autoimmune Myocarditis
- (2010) Aranzazu Cruz-Adalia et al. CIRCULATION
- The leukocyte activation antigen CD69 limits allergic asthma and skin contact hypersensitivity
- (2010) Pilar Martín et al. JOURNAL OF ALLERGY AND CLINICAL IMMUNOLOGY
- IL-23-Mediated Psoriasis-Like Epidermal Hyperplasia Is Dependent on IL-17A
- (2010) H. L. Rizzo et al. JOURNAL OF IMMUNOLOGY
- CD69 Association with Jak3/Stat5 Proteins Regulates Th17 Cell Differentiation
- (2010) P. Martin et al. MOLECULAR AND CELLULAR BIOLOGY
- Interleukin-17-Producing γδ T Cells Selectively Expand in Response to Pathogen Products and Environmental Signals
- (2009) Bruno Martin et al. IMMUNITY
- Cutting Edge: IL-23 Receptor GFP Reporter Mice Reveal Distinct Populations of IL-17-Producing Cells
- (2009) Amit Awasthi et al. JOURNAL OF IMMUNOLOGY
- IL-22 and IL-20 are key mediators of the epidermal alterations in psoriasis while IL-17 and IFN-γ are not
- (2009) Kerstin Wolk et al. JOURNAL OF MOLECULAR MEDICINE-JMM
- CD27 is a thymic determinant of the balance between interferon-γ- and interleukin 17–producing γδ T cell subsets
- (2009) Julie C Ribot et al. NATURE IMMUNOLOGY
- Regulatory mechanisms of SNAT2, an amino acid transporter, in L6 rat skeletal muscle cells by insulin, osmotic shock and amino acid deprivation
- (2008) Hitoshi Kashiwagi et al. AMINO ACIDS
- Natural agonists for aryl hydrocarbon receptor in culture medium are essential for optimal differentiation of Th17 T cells
- (2008) Marc Veldhoen et al. JOURNAL OF EXPERIMENTAL MEDICINE
Discover Peeref hubs
Discuss science. Find collaborators. Network.
Join a conversationAsk a Question. Answer a Question.
Quickly pose questions to the entire community. Debate answers and get clarity on the most important issues facing researchers.
Get Started