Does MPK4/12-HT1 function as a CO2/bicarbonate sensor to regulate the stomatal conductance under high CO2 levels?

Key message Recently, a HT1 protein has been identified which causes continuous opening of stomata because of its kinase activity. However, reversible interaction between MAP4/12 and HT1 protein acts as a CO2/bicarbonate sensor and causes the closing of stomata by inhibiting HT1 kinase activity.

Automated summary

A recent discovery of a protein called HT1 has been found to cause continuous opening of stomata due to its kinase activity. However, the reversible interaction between MAP4/12 and HT1 protein acts as a CO2/bicarbonate sensor and inhibits HT1 kinase activity, resulting in the closing of stomata.