Ficolin-1 Levels in Patients Developing Vasospasm and Cerebral Ischemia After Spontaneous Subarachnoid Hemorrhage
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Title
Ficolin-1 Levels in Patients Developing Vasospasm and Cerebral Ischemia After Spontaneous Subarachnoid Hemorrhage
Authors
Keywords
Ficolin, Subarachnoid hemorrhage, Brain ischemia, Vasospasm
Journal
MOLECULAR NEUROBIOLOGY
Volume 54, Issue 8, Pages 6572-6580
Publisher
Springer Nature
Online
2016-10-12
DOI
10.1007/s12035-016-0180-0
References
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Related references
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- Non-Synonymous Polymorphisms in the FCN1 Gene Determine Ligand-Binding Ability and Serum Levels of M-Ficolin
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- Biological variations of MASP-3 and MAp44, two splice products of the MASP1 gene involved in regulation of the complement system
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- Tethering of Ficolin-1 to cell surfaces through recognition of sialic acid by the fibrinogen-like domain
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- Genetically-Defined Deficiency of Mannose-Binding Lectin Is Associated with Protection after Experimental Stroke in Mice and Outcome in Human Stroke
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- MAp44, a Human Protein Associated with Pattern Recognition Molecules of the Complement System and Regulating the Lectin Pathway of Complement Activation
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- Characteristics and Biological Variations of M-Ficolin, a Pattern Recognition Molecule, in Plasma
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- Ficolin-1 is present in a highly mobilizable subset of human neutrophil granules and associates with the cell surface after stimulation with fMLP
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- In SituDeposition of Complement in Human Acute Brain Ischaemia
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- Expression of M-Ficolin in human monocytes and macrophages
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