Pharmacological study on the enhancing effects of U46619 on guinea pig urinary bladder smooth muscle contraction induced by acetylcholine and α,β-methylene ATP and the possible involvement of protein kinase C

Signalling pathway of U46619‐induced vascular smooth muscle contraction in mouse coronary artery

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Prostanoid TP receptor stimulation enhances contractile activities in guinea pig urinary bladder smooth muscle through activation of Ca2+ entry channels: Potential targets in the treatment of urinary bladder contractile dysfunction

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Inhibition of human prostate smooth muscle contraction by the inhibitors of protein kinase C, GF109203X, and Go6983

(2021) Ru Huang et al.   PROSTATE

Mechanisms of U46619‐induced contraction in mice intrarenal arteries

(2019) Hong Yan et al.   CLINICAL AND EXPERIMENTAL PHARMACOLOGY AND PHYSIOLOGY

Effect of distigmine on the contractile response of guinea pig urinary bladder to electrical field stimulation

(2017) Keisuke Obara et al.   EUROPEAN JOURNAL OF PHARMACOLOGY

ATP as a cotransmitter in the autonomic nervous system

(2015) Charles Kennedy   AUTONOMIC NEUROSCIENCE-BASIC & CLINICAL

Regulation of urinary bladder function by protein kinase C in physiology and pathophysiology

(2015) Joseph A. Hypolite et al.   BMC Urology

Thromboxane A2 receptor and MaxiK-channel intimate interaction supports channel trans-inhibition independent of G-protein activation

(2010) M. Li et al.   PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA

The Potent Protein Kinase C-Selective Inhibitor AEB071 (Sotrastaurin) Represents a New Class of Immunosuppressive Agents Affecting Early T-Cell Activation

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