Journal
CELL STEM CELL
Volume 16, Issue 4, Pages 341-342Publisher
CELL PRESS
DOI: 10.1016/j.stem.2015.03.010
Keywords
-
Categories
Funding
- NCI NIH HHS [R01 CA084198] Funding Source: Medline
Ask authors/readers for more resources
Understanding how genetic risk variants contribute to complex diseases is crucial for predicting disease susceptibility and developing patient-tailored therapies. In this issue of Cell Stem Cell, Young et al. (2015) dissect the function of common non-coding risk haplotypes in the SORL1 locus in the pathogenesis of sporadic Alzheimer's disease using patient-derived induced pluripotent stem cells.
Authors
I am an author on this paper
Click your name to claim this paper and add it to your profile.
Reviews
Recommended
No Data Available
Share Paper
