Synthetic miR-145 Mimic Enhances the Cytotoxic Effect of the Antiangiogenic Drug Sunitinib in Glioblastoma
Published 2015 View Full Article
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Title
Synthetic miR-145 Mimic Enhances the Cytotoxic Effect of the Antiangiogenic Drug Sunitinib in Glioblastoma
Authors
Keywords
Mir-145 mimic, Suntinib, GBM
Journal
CELL BIOCHEMISTRY AND BIOPHYSICS
Volume 72, Issue 2, Pages 551-557
Publisher
Springer Nature
Online
2015-01-07
DOI
10.1007/s12013-014-0501-8
References
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Related references
Note: Only part of the references are listed.- NEDD9, a novel target of miR-145, increases the invasiveness of glioblastoma
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- MicroRNA-145 Is Downregulated in Glial Tumors and Regulates Glioma Cell Migration by Targeting Connective Tissue Growth Factor
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- Glioblastoma Recurrence after Cediranib Therapy in Patients: Lack of "Rebound" Revascularization as Mode of Escape
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- miR-221/222 overexpession in human glioblastoma increases invasiveness by targeting the protein phosphate PTPμ
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- MicroRNAs in the Pathogenesis of Cancer
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- MGMT modulates glioblastoma angiogenesis and response to the tyrosine kinase inhibitor sunitinib
- (2010) Manik Chahal et al. NEURO-ONCOLOGY
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- MicroRNA miR-93 promotes tumor growth and angiogenesis by targeting integrin-β8
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- MicroRNA-145 Regulates OCT4, SOX2, and KLF4 and Represses Pluripotency in Human Embryonic Stem Cells
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- MicroRNA-10b is overexpressed in malignant glioma and associated with tumor invasive factors, uPAR and RhoC
- (2009) Takashi Sasayama et al. INTERNATIONAL JOURNAL OF CANCER
- Emerging functions of microRNAs in glioblastoma
- (2009) Sean Lawler et al. JOURNAL OF NEURO-ONCOLOGY
- FDA Drug Approval Summary: Bevacizumab (Avastin(R)) as Treatment of Recurrent Glioblastoma Multiforme
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- Molecular Advances of Brain Tumors in Radiation Oncology
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- Targeting of the Bmi-1 Oncogene/Stem Cell Renewal Factor by MicroRNA-128 Inhibits Glioma Proliferation and Self-Renewal
- (2008) J. Godlewski et al. CANCER RESEARCH
- Impact of Angiogenesis Inhibition by Sunitinib on Tumor Distribution of Temozolomide
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