Revisiting autoimmune lymphoproliferative syndrome caused by Fas ligand mutations
Published 2023 View Full Article
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Title
Revisiting autoimmune lymphoproliferative syndrome caused by Fas ligand mutations
Authors
Keywords
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Journal
JOURNAL OF ALLERGY AND CLINICAL IMMUNOLOGY
Volume -, Issue -, Pages -
Publisher
Elsevier BV
Online
2023-01-05
DOI
10.1016/j.jaci.2022.11.028
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Related references
Note: Only part of the references are listed.- The genetic landscape of the FAS pathway deficiencies
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- Decreased activation-induced cell death by EBV-transformed B-cells from a patient with autoimmune lymphoproliferative syndrome caused by a novel FASLG mutation
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- A novel homozygous Fas ligand mutation leads to early protein truncation, abrogation of death receptor and reverse signaling and a severe form of the autoimmune lymphoproliferative syndrome
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- Sequential decisions on FAS sequencing guided by biomarkers in patients with lymphoproliferation and autoimmune cytopenia
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- (2012) Raffick A.R. Bowen et al. CLINICAL BIOCHEMISTRY
- Autoimmune lymphoproliferative syndrome caused by a homozygous null FAS ligand (FASLG) mutation
- (2012) Aude Magerus-Chatinet et al. JOURNAL OF ALLERGY AND CLINICAL IMMUNOLOGY
- Optimization of the Additive CHARMM All-Atom Protein Force Field Targeting Improved Sampling of the Backbone ϕ, ψ and Side-Chain χ1 and χ2 Dihedral Angles
- (2012) Robert B. Best et al. Journal of Chemical Theory and Computation
- How I treat autoimmune lymphoproliferative syndrome
- (2011) V. K. Rao et al. BLOOD
- Revised diagnostic criteria and classification for the autoimmune lymphoproliferative syndrome (ALPS): report from the 2009 NIH International Workshop
- (2010) J. B. Oliveira et al. BLOOD
- Immune modulation by Fas ligand reverse signaling: lymphocyte proliferation is attenuated by the intracellular Fas ligand domain
- (2010) K. Luckerath et al. BLOOD
- Mutation of the BAFF furin cleavage site impairs B-cell homeostasis and antibody responses
- (2010) Claudia Bossen et al. EUROPEAN JOURNAL OF IMMUNOLOGY
- Using biomarkers to predict the presence of FAS mutations in patients with features of the autoimmune lymphoproliferative syndrome
- (2010) Iusta Caminha et al. JOURNAL OF ALLERGY AND CLINICAL IMMUNOLOGY
- Onset of autoimmune lymphoproliferative syndrome (ALPS) in humans as a consequence of genetic defect accumulation
- (2010) Aude Magerus-Chatinet et al. JOURNAL OF CLINICAL INVESTIGATION
- FAS-L, IL-10, and double-negative CD4-CD8- TCR / + T cells are reliable markers of autoimmune lymphoproliferative syndrome (ALPS) associated with FAS loss of function
- (2009) A. Magerus-Chatinet et al. BLOOD
- FasL cross-linking inhibits activation of human peripheral T cells
- (2009) M. Paulsen et al. INTERNATIONAL IMMUNOLOGY
- Membrane-bound Fas ligand only is essential for Fas-induced apoptosis
- (2009) Lorraine A. O’ Reilly et al. NATURE
- Fas Receptor Expression in Germinal-Center B Cells Is Essential for T and B Lymphocyte Homeostasis
- (2008) Zhenyue Hao et al. IMMUNITY
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