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Title
Orphaned mitochondria in heart failure
Authors
Keywords
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Journal
CARDIOVASCULAR RESEARCH
Volume 109, Issue 1, Pages 6-8
Publisher
Oxford University Press (OUP)
Online
2015-12-01
DOI
10.1093/cvr/cvv262
References
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Related references
Note: Only part of the references are listed.- Impaired mitochondrial network excitability in failing guinea-pig cardiomyocytes
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- Super-Suppression of Mitochondrial Reactive Oxygen Species Signaling Impairs Compensatory Autophagy in Primary Mitophagic Cardiomyopathy
- (2014) Moshi Song et al. CIRCULATION RESEARCH
- Mitochondrial reactive oxygen species production and elimination
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- Redox Signaling in Cardiac Physiology and Pathology
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- Subcellular Structures and Function of Myocytes Impaired During Heart Failure Are Restored by Cardiac Resynchronization Therapy
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- Stimulated Emission Depletion Live-Cell Super-Resolution Imaging Shows Proliferative Remodeling of T-Tubule Membrane Structures After Myocardial Infarction
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- Cardiomyocyte deletion of mitofusin-1 leads to mitochondrial fragmentation and improves tolerance to ROS-induced mitochondrial dysfunction and cell death
- (2011) Kyriakos N. Papanicolaou et al. AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY
- Mitochondrial Fusion is Essential for Organelle Function and Cardiac Homeostasis
- (2011) Yun Chen et al. CIRCULATION RESEARCH
- Mitofusin-2 Maintains Mitochondrial Structure and Contributes to Stress-Induced Permeability Transition in Cardiac Myocytes
- (2011) K. N. Papanicolaou et al. MOLECULAR AND CELLULAR BIOLOGY
- Elevated Cytosolic Na + Increases Mitochondrial Formation of Reactive Oxygen Species in Failing Cardiac Myocytes
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- Adverse Bioenergetic Consequences of Na + -Ca 2+ Exchanger–Mediated Ca 2+ Influx in Cardiac Myocytes
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