Molecular mechanisms in H 2 O 2 -induced increase in AT1 receptor gene expression in cardiac fibroblasts: A role for endogenously generated Angiotensin II
Molecular mechanisms in H 2 O 2 -induced increase in AT1 receptor gene expression in cardiac fibroblasts: A role for endogenously generated Angiotensin II
Authors
Keywords
Cardiac fibroblasts, AT1R, Oxidative stress, Angiotensin II, Collagen
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