Type I IFN Inhibits Alternative Macrophage Activation duringMycobacterium tuberculosisInfection and Leads to Enhanced Protection in the Absence of IFN-γ Signaling
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Title
Type I IFN Inhibits Alternative Macrophage Activation duringMycobacterium tuberculosisInfection and Leads to Enhanced Protection in the Absence of IFN-γ Signaling
Authors
Keywords
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Journal
JOURNAL OF IMMUNOLOGY
Volume 197, Issue 12, Pages 4714-4726
Publisher
The American Association of Immunologists
Online
2016-11-15
DOI
10.4049/jimmunol.1600584
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Note: Only part of the references are listed.- Cytokine and lipid mediator networks in tuberculosis
- (2015) Katrin D. Mayer-Barber et al. IMMUNOLOGICAL REVIEWS
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- (2014) Finlay W. McNab et al. JOURNAL OF IMMUNOLOGY
- The IL-13/IL-4Rαaxis is involved in tuberculosis-associated pathology
- (2014) Lisa Heitmann et al. JOURNAL OF PATHOLOGY
- Host-directed therapy of tuberculosis based on interleukin-1 and type I interferon crosstalk
- (2014) Katrin D. Mayer-Barber et al. NATURE
- The Immune Response in Tuberculosis
- (2013) Anne O'Garra et al. Annual Review of Immunology
- Inhibition of the type I immune responses of human monocytes by IFN-α and IFN-β
- (2013) Roelof A. de Paus et al. CYTOKINE
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- (2013) H. I. Bax et al. JOURNAL OF CLINICAL IMMUNOLOGY
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- (2013) F. W. McNab et al. JOURNAL OF IMMUNOLOGY
- Influenza A Virus Impairs Control of Mycobacterium tuberculosis Coinfection Through a Type I Interferon Receptor–Dependent Pathway
- (2013) Paul S. Redford et al. JOURNAL OF INFECTIOUS DISEASES
- Mycobacterium tuberculosis Strains Are Differentially Recognized by TLRs with an Impact on the Immune Response
- (2013) Jenny Carmona et al. PLoS One
- Type I Interferon Suppresses Type II Interferon-Triggered Human Anti-Mycobacterial Responses
- (2013) R. M. B. Teles et al. SCIENCE
- Arginase-1 expression in granulomas of tuberculosis patients: Figure 1
- (2012) Ana P. Pessanha et al. FEMS IMMUNOLOGY AND MEDICAL MICROBIOLOGY
- Nitric Oxide Increases Susceptibility of Toll-like Receptor-Activated Macrophages to Spreading Listeria monocytogenes
- (2012) Caroline Cole et al. IMMUNITY
- Dynamic Roles of Type I and Type II IFNs in Early Infection with Mycobacterium tuberculosis
- (2012) L. Desvignes et al. JOURNAL OF IMMUNOLOGY
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- (2012) Jacqueline M. Cliff et al. JOURNAL OF INFECTIOUS DISEASES
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- (2012) Bibhuti B Mishra et al. NATURE IMMUNOLOGY
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- (2012) Tom H. M. Ottenhoff et al. PLoS One
- Detectable Changes in The Blood Transcriptome Are Present after Two Weeks of Antituberculosis Therapy
- (2012) Chloe I. Bloom et al. PLoS One
- Innate Immune Recognition ofMycobacterium tuberculosis
- (2011) Johanneke Kleinnijenhuis et al. Clinical & Developmental Immunology
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- (2011) Julius A. Potian et al. JOURNAL OF EXPERIMENTAL MEDICINE
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- Human gene expression profiles of susceptibility and resistance in tuberculosis
- (2010) J Maertzdorf et al. GENES AND IMMUNITY
- Intranasal Poly-IC treatment exacerbates tuberculosis in mice through the pulmonary recruitment of a pathogen-permissive monocyte/macrophage population
- (2010) Lis R.V. Antonelli et al. JOURNAL OF CLINICAL INVESTIGATION
- An interferon-inducible neutrophil-driven blood transcriptional signature in human tuberculosis
- (2010) Matthew P. R. Berry et al. NATURE
- Arginine Usage in Mycobacteria-Infected Macrophages Depends on Autocrine-Paracrine Cytokine Signaling
- (2010) J. E. Qualls et al. Science Signaling
- Cell-Mediated Immune Responses in Tuberculosis
- (2009) Andrea M. Cooper Annual Review of Immunology
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- (2008) Karim C El Kasmi et al. NATURE IMMUNOLOGY
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