4.5 Article

Importance of keratinized mucosa around dental implants: Consensus report of group 1 of the DGI/SEPA/Osteology Workshop

Journal

CLINICAL ORAL IMPLANTS RESEARCH
Volume 33, Issue -, Pages 47-55

Publisher

WILEY
DOI: 10.1111/clr.13956

Keywords

autogenous grafts; connective tissue attachment; dental implants; keratinized mucosa; oral epithelium; soft-tissue substitutes; xenogeneic grafts

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This study examines the relevance of keratinized peri-implant mucosa (KPIM) in maintaining the health and stability of peri-implant tissues and explores surgical interventions and grafting materials for increasing KPIM dimensions when it is lacking. The findings suggest that reduced KPIM width is associated with various negative outcomes and that surgical interventions using autogenous grafts or substitutes can be effective.
Objectives To assess the literature on (i) the relevance of the presence of a minimum dimension of keratinized peri-implant mucosa (KPIM) to maintain the health and stability of peri-implant tissues, and; (ii) the surgical interventions and grafting materials used for augmenting the dimensions of the KPIM when there is a minimal amount or absence of it. Material & Methods Two systematic reviews complemented by expert opinion from workshop group participants served as the basis of the consensus statements, implications for clinical practice and future research, and were approved in plenary session by all workshop participants. Results Thirty-four consensus statements, eight implications for clinical practice, and 13 implications for future research were discussed and agreed upon. There is no consistent data on the incidence of peri-implant mucositis relative to the presence or absence of KPIM. However, reduced KPIM width is associated with increased biofilm accumulation, soft-tissue inflammation, greater patient discomfort, mucosal recession, marginal bone loss and an increased prevalence of peri-implantitis. Free gingival autogenous grafts were considered the standard of care surgical intervention to effectively increase the width of KPIM. However, substitutes of xenogeneic origin may be an alternative to autogenous tissues, since similar results when compared to connective tissue grafts were reported. Conclusion Presence of a minimum width of KPIM should be assessed routinely in patients with implant supported restorations, and when associated with pathological changes in the peri-implant mucosa, its dimensions may be surgically increased using autogenous grafts or soft-tissue substitutes with evidence of proven efficacy.

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