Loss of Integrin α9β1 on Tumor Keratinocytes Enhances the Stromal Vasculature and Growth of Cutaneous Tumors

Clinically relevant aberrant Filip1l DNA methylation detected in a murine model of cutaneous squamous cell carcinoma

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Integrin α4β1 is required for IL ‐1α‐ and Nrf2‐dependent, Cox‐2 induction in fibroblasts, supporting a mechanism that suppresses α‐SMA expression

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Integrin α3β1 on tumor keratinocytes is essential to maintain tumor growth and promotes a tumor-supportive keratinocyte secretome.

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UVB induces cutaneous squamous cell carcinoma progression by de novo ID4 methylation via methylation regulating enzymes

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Keratinocyte integrin α3β1 promotes secretion of IL-1α to effect paracrine regulation of fibroblast gene expression and differentiation

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Integrin α9 depletion promotes β‐catenin degradation to suppress triple‐negative breast cancer tumor growth and metastasis

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Methylation profiling identifies two subclasses of squamous cell carcinoma related to distinct cells of origin

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Suppression of integrin α3β1 by α9β1 in the epidermis controls the paracrine resolution of wound angiogenesis

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Cell Adhesion Mediated by VCAM-ITG 9 Interactions Enables Lymphatic Development

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EphB2 Promotes Progression of Cutaneous Squamous Cell Carcinoma

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Comprehensive genomic characterization of head and neck squamous cell carcinomas

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Evolution of metastasis revealed by mutational landscapes of chemically induced skin cancers

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Systematic pan-cancer analysis of tumour purity

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Integrin α9 gene promoter is hypermethylated and downregulated in nasopharyngeal carcinoma

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Gene Expression Profiling of the Leading Edge of Cutaneous Squamous Cell Carcinoma: IL-24-Driven MMP-7

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Integrative Analysis of Complex Cancer Genomics and Clinical Profiles Using the cBioPortal

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Integrin α9β1 promotes malignant tumor growth and metastasis by potentiating epithelial-mesenchymal transition

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Loss of integrin  3 prevents skin tumor formation by promoting epidermal turnover and depletion of slow-cycling cells

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The cBio Cancer Genomics Portal: An Open Platform for Exploring Multidimensional Cancer Genomics Data: Figure 1.

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Integrin alpha9 (ITGA9) expression and epigenetic silencing in human breast tumors

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GISTIC2.0 facilitates sensitive and confident localization of the targets of focal somatic copy-number alteration in human cancers

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Positional differences in the wound transcriptome of skin and oral mucosa

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Integrin-α9 Is Required for Fibronectin Matrix Assembly during Lymphatic Valve Morphogenesis

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 3 1 integrin in epidermis promotes wound angiogenesis and keratinocyte-to-endothelial-cell crosstalk through the induction of MRP3

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Systematic RNA interference reveals that oncogenic KRAS-driven cancers require TBK1

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Multi-stage chemical carcinogenesis in mouse skin: Fundamentals and applications

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An Immortalization-Dependent Switch in Integrin Function Up-regulates MMP-9 to Enhance Tumor Cell Invasion

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Induction of pluripotent stem cells by defined factors is greatly improved by small-molecule compounds

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Isolation and short-term culture of primary keratinocytes, hair follicle populations and dermal cells from newborn mice and keratinocytes from adult mice for in vitro analysis and for grafting to immunodeficient mice

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Cancer as an overhealing wound: an old hypothesis revisited

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