N-acetylcysteine Can Induce Massive Oxidative Stress, Resulting in Cell Death with Apoptotic Features in Human Leukemia Cells
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Title
N-acetylcysteine Can Induce Massive Oxidative Stress, Resulting in Cell Death with Apoptotic Features in Human Leukemia Cells
Authors
Keywords
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Journal
INTERNATIONAL JOURNAL OF MOLECULAR SCIENCES
Volume 22, Issue 23, Pages 12635
Publisher
MDPI AG
Online
2021-11-30
DOI
10.3390/ijms222312635
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- (2020) Sofia K. Georgiou-Siafis et al. EUROPEAN JOURNAL OF PHARMACOLOGY
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- (2020) Ankush Prasad et al. Frontiers in Physiology
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- (2019) P. Mlejnek et al. ENVIRONMENTAL TOXICOLOGY AND PHARMACOLOGY
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- Activation of apoptosis signalling pathways by reactive oxygen species
- (2016) Maureen Redza-Dutordoir et al. BIOCHIMICA ET BIOPHYSICA ACTA-MOLECULAR CELL RESEARCH
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- Loss of mitochondrial transmembrane potential and glutathione depletion are not sufficient to account for induction of apoptosis by carbonyl cyanide 4-(trifluoromethoxy)phenylhydrazone in human leukemia K562 cells
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- N-acetyl-L-cysteine enhances fisetin-induced cytotoxicity via induction of ROS-independent apoptosis in human colonic cancer cells
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- Existing and potential therapeutic uses for N-acetylcysteine: The need for conversion to intracellular glutathione for antioxidant benefits
- (2013) Gordon F. Rushworth et al. PHARMACOLOGY & THERAPEUTICS
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- (2010) Jie Zheng et al. CANCER LETTERS
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- (2009) Srabanti Rakshit et al. APOPTOSIS
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- (2009) Eva Kriegova et al. BMC MOLECULAR BIOLOGY
- Regulation of Autophagy by Reactive Oxygen Species (ROS): Implications for Cancer Progression and Treatment
- (2008) Meghan B. Azad et al. ANTIOXIDANTS & REDOX SIGNALING
- Structure, regulation and evolution of Nox-family NADPH oxidases that produce reactive oxygen species
- (2008) Hideki Sumimoto FEBS Journal
- Serine protease inhibitorsN-α-Tosyl-L-Lysinyl-Chloromethylketone (TLCK) andN-Tosyl-L-Phenylalaninyl-Chloromethylketone (TPCK) are potent inhibitors of activated caspase proteases
- (2007) Ivo Frydrych et al. JOURNAL OF CELLULAR BIOCHEMISTRY
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