S100A9 expressed in ER−PgR− breast cancers induces inflammatory cytokines and is associated with an impaired overall survival
Published 2015 View Full Article
- Home
- Publications
- Publication Search
- Publication Details
Title
S100A9 expressed in ER−PgR− breast cancers induces inflammatory cytokines and is associated with an impaired overall survival
Authors
Keywords
-
Journal
BRITISH JOURNAL OF CANCER
Volume 113, Issue 8, Pages 1234-1243
Publisher
Springer Nature
Online
2015-10-09
DOI
10.1038/bjc.2015.346
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- S100A9 and EGFR gene signatures predict disease progression in muscle invasive bladder cancer patients after chemotherapy
- (2014) W. T. Kim et al. ANNALS OF ONCOLOGY
- Intracellular Expression of Inflammatory Proteins S100A8 and S100A9 Leads to Epithelial-mesenchymal Transition and Attenuated Aggressivity of Breast Cancer Cells
- (2014) Kevin Cormier et al. Anti-Cancer Agents in Medicinal Chemistry
- High density of S100A9 positive inflammatory cells in prostate cancer stroma is associated with poor outcome
- (2014) Viktor Tidehag et al. EUROPEAN JOURNAL OF CANCER
- Proinflammatory S100 Proteins Regulate the Accumulation of Myeloid-Derived Suppressor Cells
- (2014) P. Sinha et al. JOURNAL OF IMMUNOLOGY
- ID1 Promotes Breast Cancer Metastasis by S100A9 Regulation
- (2014) K. Gumireddy et al. MOLECULAR CANCER RESEARCH
- RAGE-binding S100A8/A9 promotes the migration and invasion of human breast cancer cells through actin polymerization and epithelial–mesenchymal transition
- (2013) Chonggao Yin et al. BREAST CANCER RESEARCH AND TREATMENT
- Human S100A9 Protein Is Stabilized by Inflammatory Stimuli via the Formation of Proteolytically-Resistant Homodimers
- (2013) Matteo Riva et al. PLoS One
- Common Interactions between S100A4 and S100A9 Defined by a Novel Chemical Probe
- (2013) Per Björk et al. PLoS One
- The presence of tumor associated macrophages in tumor stroma as a prognostic marker for breast cancer patients
- (2012) Catharina Medrek et al. BMC CANCER
- A CXCL1 Paracrine Network Links Cancer Chemoresistance and Metastasis
- (2012) Swarnali Acharyya et al. CELL
- Induction of nuclear factor-κB responses by the S100A9 protein is Toll-like receptor-4-dependent
- (2012) Matteo Riva et al. IMMUNOLOGY
- Interleukin-6 Induces S100A9 Expression in Colonic Epithelial Cells through STAT3 Activation in Experimental Ulcerative Colitis
- (2012) Min Jeoung Lee et al. PLoS One
- Expression of ghrelin is correlated to a favorable outcome in invasive breast cancer
- (2011) Malin Grönberg et al. ACTA ONCOLOGICA
- Human tumors instigate granulin-expressing hematopoietic cells that promote malignancy by activating stromal fibroblasts in mice
- (2011) Moshe Elkabets et al. JOURNAL OF CLINICAL INVESTIGATION
- Classification and prognosis of invasive breast cancer: from morphology to molecular taxonomy
- (2010) Stuart J Schnitt MODERN PATHOLOGY
- Myeloid-Derived Suppressor Cells: Linking Inflammation and Cancer
- (2009) S. Ostrand-Rosenberg et al. JOURNAL OF IMMUNOLOGY
- Identification of Human S100A9 as a Novel Target for Treatment of Autoimmune Disease via Binding to Quinoline-3-Carboxamides
- (2009) Per Björk et al. PLOS BIOLOGY
- S100A8 and S100A9 Overexpression Is Associated with Poor Pathological Parameters in Invasive Ductal Carcinoma of the Breast
- (2008) Kazumori Arai et al. CURRENT CANCER DRUG TARGETS
- Protein Profiling of Human Breast Tumor Cells Identifies Novel Biomarkers Associated with Molecular Subtypes
- (2008) Anthony Gonçalves et al. MOLECULAR & CELLULAR PROTEOMICS
- Global Gene Expression Profiling Unveils S100A8/A9 as Candidate Markers in H-Ras-Mediated Human Breast Epithelial Cell Invasion
- (2008) A. Moon et al. MOLECULAR CANCER RESEARCH
Add your recorded webinar
Do you already have a recorded webinar? Grow your audience and get more views by easily listing your recording on Peeref.
Upload NowCreate your own webinar
Interested in hosting your own webinar? Check the schedule and propose your idea to the Peeref Content Team.
Create Now