Mitochondrial impairments contribute to Spinocerebellar ataxia type 1 progression and can be ameliorated by the mitochondria-targeted antioxidant MitoQ
Mitochondrial impairments contribute to Spinocerebellar ataxia type 1 progression and can be ameliorated by the mitochondria-targeted antioxidant MitoQ
Authors
Keywords
Spinocerebellar ataxia type 1, Purkinje cells, Mitochondria, Oxidative stress, Electron transport chain (ETC) activities, Neurodegeneration, Antioxidant MitoQ
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