Upregulation of oxidative stress gene markers during SARS-COV-2 viral infection

Global pandemics interconnected — obesity, impaired metabolic health and COVID-19

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Calprotectin in Lung Diseases

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A useful and sensitive marker in the prediction of COVID-19 and disease severity: Thiol

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SARS-CoV-2 Switches ‘on’ MAPK and NFκB Signaling via the Reduction of Nuclear DUSP1 and DUSP5 Expression

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SARS-CoV-2 Cell Entry Depends on ACE2 and TMPRSS2 and Is Blocked by a Clinically Proven Protease Inhibitor

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Elevated calprotectin and abnormal myeloid cell subsets discriminate severe from mild COVID-19

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Transcriptional and proteomic insights into the host response in fatal COVID-19 cases

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SARS-CoV2-mediated suppression of NRF2-signaling reveals potent antiviral and anti-inflammatory activity of 4-octyl-itaconate and dimethyl fumarate

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Induction of alarmin S100A8/A9 mediates activation of aberrant neutrophils in the pathogenesis of COVID-19

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SARS-CoV-2 and Other Respiratory Viruses: What Does Oxidative Stress Have to Do with It?

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Oxidative cross-linking of calprotectin occurs in vivo, altering its structure and susceptibility to proteolysis

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High Level of Neutrophil Extracellular Traps Correlates With Poor Prognosis of Severe Influenza A Infection

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Implications of oxidative stress on viral pathogenesis

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NADPH Oxidase 1 Is Associated with Altered Host Survival and T Cell Phenotypes after Influenza A Virus Infection in Mice

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Redox-Active Protein Thioredoxin-1 Administration Ameliorates Influenza A Virus (H1N1)-Induced Acute Lung Injury in Mice

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18S rRNA is a reliable normalisation gene for real time PCR based on influenza virus infected cells

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Viral-mediated Inhibition of Antioxidant Enzymes Contributes to the Pathogenesis of Severe Respiratory Syncytial Virus Bronchiolitis

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Inhibitory effect of melatonin on lung oxidative stress induced by respiratory syncytial virus infection in mice

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Gene Expression Signatures Diagnose Influenza and Other Symptomatic Respiratory Viral Infections in Humans

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S100A8/A9 induces autophagy and apoptosis via ROS-mediated cross-talk between mitochondria and lysosomes that involves BNIP3

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Nrf2:INrf2 (Keap1) signaling in oxidative stress

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Modulation of TNF- -converting enzyme by the spike protein of SARS-CoV and ACE2 induces TNF-  production and facilitates viral entry

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