Chronic Diclofenac Exposure Increases Mitochondrial Oxidative Stress, Inflammatory Mediators, and Cardiac Dysfunction
Published 2021 View Full Article
- Home
- Publications
- Publication Search
- Publication Details
Title
Chronic Diclofenac Exposure Increases Mitochondrial Oxidative Stress, Inflammatory Mediators, and Cardiac Dysfunction
Authors
Keywords
-
Journal
CARDIOVASCULAR DRUGS AND THERAPY
Volume -, Issue -, Pages -
Publisher
Springer Science and Business Media LLC
Online
2021-09-09
DOI
10.1007/s10557-021-07253-4
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- Suppression of inflammation and fibrosis using soluble epoxide hydrolase inhibitors enhances cardiac stem cell-based therapy
- (2020) Padmini Sirish et al. Stem Cells Translational Medicine
- Dose-dependency of the cardiovascular risks of non-steroidal anti-inflammatory drugs
- (2019) Surur Ali Ahmed et al. INFLAMMOPHARMACOLOGY
- Mitochondrial dysfunction and oxidative stress in heart disease
- (2019) Jessica N. Peoples et al. EXPERIMENTAL AND MOLECULAR MEDICINE
- Exceeding the daily dosing limit of nonsteroidal anti-inflammatory drugs among ibuprofen users
- (2018) David W. Kaufman et al. PHARMACOEPIDEMIOLOGY AND DRUG SAFETY
- Mitochondrial oxidative stress activates COX-2/mPGES-1/PGE2 cascade induced by albumin in renal proximal tubular cells
- (2018) Yibo Zhuang et al. Oncotarget
- A Comprehensive Review of Non-Steroidal Anti-Inflammatory Drug Use in The Elderly
- (2018) Supakanya Wongrakpanich et al. Aging and Disease
- Synopsis of arachidonic acid metabolism: A review
- (2018) Violette Said Hanna et al. Journal of Advanced Research
- Cardiac-specific Conditional Knockout of the 18-kDa Mitochondrial Translocator Protein Protects from Pressure Overload Induced Heart Failure
- (2018) Phung N. Thai et al. Scientific Reports
- Diclofenac use and cardiovascular risks: series of nationwide cohort studies
- (2018) Morten Schmidt et al. BMJ-British Medical Journal
- Cardiomyocyte Inflammasome Signaling in Cardiomyopathies and Atrial Fibrillation: Mechanisms and Potential Therapeutic Implications
- (2018) Gong Chen et al. Frontiers in Physiology
- Mitochondrial Membrane Potential Regulates Nuclear Gene Expression in Macrophages Exposed to Prostaglandin E2
- (2018) David E. Sanin et al. IMMUNITY
- Pharmacological inhibition of soluble epoxide hydrolase or genetic deletion reduces diclofenac-induced gastric ulcers
- (2017) Sumanta Kumar Goswami et al. LIFE SCIENCES
- Risk of acute myocardial infarction with NSAIDs in real world use: bayesian meta-analysis of individual patient data
- (2017) Michèle Bally et al. BMJ-British Medical Journal
- Risk of acute myocardial infarction with NSAIDs in real world use: bayesian meta-analysis of individual patient data
- (2017) Michèle Bally et al. BMJ-British Medical Journal
- Molecular Mechanisms and New Treatment Paradigm for Atrial Fibrillation
- (2016) Padmini Sirish et al. Circulation-Arrhythmia and Electrophysiology
- Diclofenac induces proteasome and mitochondrial dysfunction in murine cardiomyocytes and hearts
- (2016) Rajeshwary Ghosh et al. INTERNATIONAL JOURNAL OF CARDIOLOGY
- Mitochondrial function as a therapeutic target in heart failure
- (2016) David A. Brown et al. Nature Reviews Cardiology
- Key Role of ROS in the Process of 15-Lipoxygenase/15-Hydroxyeicosatetraenoiccid-Induced Pulmonary Vascular Remodeling in Hypoxia Pulmonary Hypertension
- (2016) Qian Li et al. PLoS One
- Mitochondrial toxicity of diclofenac and its metabolites via inhibition of oxidative phosphorylation (ATP synthesis) in rat liver mitochondria: Possible role in drug induced liver injury (DILI)
- (2016) Muzeeb Syed et al. TOXICOLOGY IN VITRO
- Calcium and ROS: A mutual interplay
- (2015) Agnes Görlach et al. Redox Biology
- Effects of non-steroidal anti-inflammatory drug (NSAID) diclofenac exposure in mussel Mytilus galloprovincialis
- (2014) Maria Gonzalez-Rey et al. AQUATIC TOXICOLOGY
- Cardiac Mitochondria and Reactive Oxygen Species Generation
- (2014) Yeong-Renn Chen et al. CIRCULATION RESEARCH
- Mechanisms of Altered Ca 2+ Handling in Heart Failure
- (2013) Min Luo et al. CIRCULATION RESEARCH
- Unique mechanistic insights into the beneficial effects of soluble epoxide hydrolase inhibitors in the prevention of cardiac fibrosis
- (2013) P. Sirish et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Adrenergic signaling and oxidative stress: a role for sirtuins?
- (2013) Graziamaria Corbi et al. Frontiers in Physiology
- Regulation of ATP production by mitochondrial Ca2+
- (2012) Andrei I. Tarasov et al. CELL CALCIUM
- Mitochondrial Oxidative Stress Mediates Angiotensin II–Induced Cardiac Hypertrophy and Gαq Overexpression–Induced Heart Failure
- (2011) Dao-Fu Dai et al. CIRCULATION RESEARCH
- Calcium Signaling in Cardiac Myocytes
- (2011) C. J. Fearnley et al. Cold Spring Harbor Perspectives in Biology
- Scutellarin alleviates interstitial fibrosis and cardiac dysfunction of infarct rats by inhibiting TGFβ1 expression and activation of p38-MAPK and ERK1/2
- (2010) Zhenwei Pan et al. BRITISH JOURNAL OF PHARMACOLOGY
- Diclofenac, a Non-steroidal Anti-inflammatory Drug, Inhibits L-type Ca2+ Channels in Neonatal Rat Ventricular Cardiomyocytes
- (2010) Oleg V. Yarishkin et al. KOREAN JOURNAL OF PHYSIOLOGY & PHARMACOLOGY
- Regulation of mitochondrial dehydrogenases by calcium ions
- (2009) Richard M. Denton BIOCHIMICA ET BIOPHYSICA ACTA-BIOENERGETICS
- Mitofusin 2 tethers endoplasmic reticulum to mitochondria
- (2008) Olga Martins de Brito et al. NATURE
Find Funding. Review Successful Grants.
Explore over 25,000 new funding opportunities and over 6,000,000 successful grants.
ExploreAsk a Question. Answer a Question.
Quickly pose questions to the entire community. Debate answers and get clarity on the most important issues facing researchers.
Get Started