Global knockdown of glutamate decarboxylase 67 elicits emotional abnormality in mice
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Title
Global knockdown of glutamate decarboxylase 67 elicits emotional abnormality in mice
Authors
Keywords
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Journal
Molecular Brain
Volume 14, Issue 1, Pages -
Publisher
Springer Science and Business Media LLC
Online
2021-01-07
DOI
10.1186/s13041-020-00713-2
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Note: Only part of the references are listed.- Glutamic acid decarboxylase 67 haplodeficiency in mice: consequences of postweaning social isolation on behavior and changes in brain neurochemical systems
- (2020) Sven Nullmeier et al. Brain Structure & Function
- Inborn vs. acquired anxiety in cross-breeding and cross-fostering HAB/LAB mice bred for extremes in anxiety-related behavior.
- (2019) Natalia Y. Chekmareva et al. BEHAVIORAL NEUROSCIENCE
- Loss of Glutamate Decarboxylase 67 in Somatostatin-Expressing Neurons Leads to Anxiety-Like Behavior and Alteration in the Akt/GSK3β Signaling Pathway
- (2019) Shigeo Miyata et al. Frontiers in Behavioral Neuroscience
- Sustained Molecular Pathology Across Episodes and Remission in Major Depressive Disorder
- (2018) Enzo Scifo et al. BIOLOGICAL PSYCHIATRY
- A novel homozygous mutation in GAD1 gene described in a schizophrenic patient impairs activity and dimerization of GAD67 enzyme
- (2018) Chiara Magri et al. Scientific Reports
- GAD1 alternative transcripts and DNA methylation in human prefrontal cortex and hippocampus in brain development, schizophrenia
- (2017) R Tao et al. MOLECULAR PSYCHIATRY
- Neuropsychiatric Phenotypes Produced by GABA Reduction in Mouse Cortex and Hippocampus
- (2017) Stefan M Kolata et al. NEUROPSYCHOPHARMACOLOGY
- Exome sequencing in schizophrenic patients with high levels of homozygosity identifies novel and extremely rare mutations in the GABA/glutamatergic pathways
- (2017) Edoardo Giacopuzzi et al. PLoS One
- Tet-On Systems For Doxycycline-inducible Gene Expression
- (2016) Atze T. Das et al. CURRENT GENE THERAPY
- Blood Transcriptomic Markers in Patients with Late-Onset Major Depressive Disorder
- (2016) Shigeo Miyata et al. PLoS One
- Cortical Gene Expression After a Conditional Knockout of 67 kDa Glutamic Acid Decarboxylase in Parvalbumin Neurons
- (2016) Danko Georgiev et al. SCHIZOPHRENIA BULLETIN
- Inhibition of parvalbumin-expressing interneurons results in complex behavioral changes
- (2015) J A Brown et al. MOLECULAR PSYCHIATRY
- Glutamate Decarboxylase 67 Deficiency in a Subset of GABAergic Neurons Induces Schizophrenia-Related Phenotypes
- (2015) Kazuyuki Fujihara et al. NEUROPSYCHOPHARMACOLOGY
- Distribution of glutamatergic, GABAergic, and glycinergic neurons in the auditory pathways of macaque monkeys
- (2015) T. Ito et al. NEUROSCIENCE
- GAD65/GAD67 double knockout mice exhibit intermediate severity in both cleft palate and omphalocele compared with GAD67 knockout and VGAT knockout mice
- (2015) T. Kakizaki et al. NEUROSCIENCE
- Transcriptional regulation of GAD1 GABA synthesis gene in the prefrontal cortex of subjects with schizophrenia
- (2015) Amanda C. Mitchell et al. SCHIZOPHRENIA RESEARCH
- Contribution of parvalbumin and somatostatin-expressing GABAergic neurons to slow oscillations and the balance in beta-gamma oscillations across cortical layers
- (2015) Toshinobu Kuki et al. Frontiers in Neural Circuits
- Altered Markers of Cortical γ-Aminobutyric Acid Neuronal Activity in Schizophrenia
- (2015) Sohei Kimoto et al. JAMA Psychiatry
- Glutamic acid decarboxylase 67 haplodeficiency impairs social behavior in mice
- (2014) K. V. Sandhu et al. GENES BRAIN AND BEHAVIOR
- Are there depression and anxiety genetic markers and mutations? A systematic review
- (2014) Sally França Lacerda-Pinheiro et al. JOURNAL OF AFFECTIVE DISORDERS
- Acoustic Experience Alters the Aged Auditory System
- (2012) Jeremy G. Turner et al. EAR AND HEARING
- Expanding the Repertoire of Optogenetically Targeted Cells with an Enhanced Gene Expression System
- (2012) Kenji F. Tanaka et al. Cell Reports
- Cortical Deficits of Glutamic Acid Decarboxylase 67 Expression in Schizophrenia: Clinical, Protein, and Cell Type-Specific Features
- (2011) Allison A. Curley et al. AMERICAN JOURNAL OF PSYCHIATRY
- Flexible Accelerated STOP Tetracycline Operator-Knockin (FAST): A Versatile and Efficient New Gene Modulating System
- (2010) Kenji F. Tanaka et al. BIOLOGICAL PSYCHIATRY
- GAD67-GFP knock-in mice have normal sleep–wake patterns and sleep homeostasis
- (2010) Lichao Chen et al. NEUROREPORT
- Low accumulation of drebrin at glutamatergic postsynaptic sites on GABAergic neurons
- (2010) K. Hanamura et al. NEUROSCIENCE
- Genetic association studies of glutamate, GABA and related genes in schizophrenia and bipolar disorder: A decade of advance
- (2010) Suat Ying Tan Cherlyn et al. NEUROSCIENCE AND BIOBEHAVIORAL REVIEWS
- A heterozygous deletion in the glutamate decarboxylase 67 gene enhances maternal and fetal stress vulnerability
- (2010) Taku Uchida et al. NEUROSCIENCE RESEARCH
- Reduced level of glutamic acid decarboxylase-67 kDa in the prefrontal cortex in major depression
- (2009) Beata Karolewicz et al. INTERNATIONAL JOURNAL OF NEUROPSYCHOPHARMACOLOGY
- Fluorescent labeling of both GABAergic and glycinergic neurons in vesicular GABA transporter (VGAT)–Venus transgenic mouse
- (2009) Y. Wang et al. NEUROSCIENCE
- GABA and synaptic inhibition of mouse cerebellum lacking glutamate decarboxylase 67
- (2008) Kunihiko Obata et al. BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS
- Age-related changes in GAD levels in the central auditory system of the rat
- (2008) Jana Burianova et al. EXPERIMENTAL GERONTOLOGY
- Deletion of GAD67 in dopamine receptor-1 expressing cells causes specific motor deficits
- (2008) Carrie L. Heusner et al. GENESIS
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