Inositol Trisphosphate Receptors and Nuclear Calcium in Atrial Fibrillation

CaMKIIδC Drives Early Adaptive Ca 2+ Change and Late Eccentric Cardiac Hypertrophy

(2020) Senka Ljubojevic-Holzer et al.   CIRCULATION RESEARCH

Dominant Frequency Increase Rate Predicts Transition from Paroxysmal to Long-Term Persistent Atrial Fibrillation

(2014) Raphael P. Martins et al.   CIRCULATION

Nuclear Calcium Transients

(2014) Paula A. da Costa Martins et al.   CIRCULATION

HDAC4: mechanism of regulation and biological functions

(2014) Zhengke Wang et al.   Epigenomics

Cellular and Molecular Mechanisms of Atrial Arrhythmogenesis in Patients With Paroxysmal Atrial Fibrillation

(2013) Niels Voigt et al.   CIRCULATION

Transient Receptor Potential Canonical-3 Channel–Dependent Fibroblast Regulation in Atrial Fibrillation

(2012) Masahide Harada et al.   CIRCULATION

Nuclear inositol 1,4,5-trisphosphate is a necessary and conserved signal for the induction of both pathological and physiological cardiomyocyte hypertrophy

(2012) Lilian A.M. Arantes et al.   JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY

Alterations of atrial Ca2+ handling as cause and consequence of atrial fibrillation

(2011) M. Greiser et al.   CARDIOVASCULAR RESEARCH

Location Matters

(2011) Shikha Mishra et al.   CIRCULATION RESEARCH

CaMKII in myocardial hypertrophy and heart failure

(2011) Mark E. Anderson et al.   JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY

Atrial Remodeling and Atrial Fibrillation

(2008) Stanley Nattel et al.   Circulation-Arrhythmia and Electrophysiology

Complex Actions of 2-Aminoethyldiphenyl Borate on Store-operated Calcium Entry

(2008) Wayne I. DeHaven et al.   JOURNAL OF BIOLOGICAL CHEMISTRY

IP3 receptor-dependent Ca2+ release modulates excitation-contraction coupling in rabbit ventricular myocytes

(2007) Timothy L. Domeier et al.   AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY