Commentary: SARS-CoV-2 Cell Entry Depends on ACE2 and TMPRSS2 and Is Blocked by a Clinically Proven Protease Inhibitor

A pneumonia outbreak associated with a new coronavirus of probable bat origin

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Structural basis for the recognition of the SARS-CoV-2 by full-length human ACE2

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SARS-CoV-2 Cell Entry Depends on ACE2 and TMPRSS2 and Is Blocked by a Clinically Proven Protease Inhibitor

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Expression of ACE2, the SARS-CoV-2 Receptor, and TMPRSS2 in Prostate Epithelial Cells

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Organ-protective effect of angiotensin-converting enzyme 2 and its effect on the prognosis of COVID-19

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Cell entry mechanisms of SARS-CoV-2

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Clinical Characteristics and Results of Semen Tests Among Men With Coronavirus Disease 2019

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Inflammation-Induced Oxidative Stress Mediates Gene Fusion Formation in Prostate Cancer

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Epigenetic impact of infection on carcinogenesis: mechanisms and applications

(2016) Naoko Hattori et al.   Genome Medicine

Role of Proprotein Convertases in Prostate Cancer Progression

(2015) Frédéric Couture et al.   NEOPLASIA

Androgens and androgen receptor signaling in prostate tumorigenesis

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Efficient Activation of the Severe Acute Respiratory Syndrome Coronavirus Spike Protein by the Transmembrane Protease TMPRSS2

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